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Regulation of biglycan gene expression by transforming growth factor-beta requires MKK6-p38 mitogen-activated protein Kinase signaling downstream of Smad signaling.
Ungefroren H, Lenschow W, Chen WB, Faendrich F, Kalthoff H. Ungefroren H, et al. J Biol Chem. 2003 Mar 28;278(13):11041-9. doi: 10.1074/jbc.M300035200. Epub 2003 Jan 20. J Biol Chem. 2003. PMID: 12538652 Free article.
-B., Lenschow, W., Tiede, K., Fischer, J. W., Kalthoff, H., and Ungefroren, H. (2002) J. Biol. Chem. 277, 36118-36128). Here, we present evidence that the ability of TGF-beta 1 to induce BGN mRNA, in addition to Smads, requires p38 MAPK signaling, because 1) …
-B., Lenschow, W., Tiede, K., Fischer, J. W., Kalthoff, H., and Ungefroren, H. (2002) J. Biol. Chem. 277, 36118-36128). …
Transforming growth factor-beta (TGF-beta) type I receptor/ALK5-dependent activation of the GADD45beta gene mediates the induction of biglycan expression by TGF-beta.
Ungefroren H, Groth S, Ruhnke M, Kalthoff H, Fändrich F. Ungefroren H, et al. J Biol Chem. 2005 Jan 28;280(4):2644-52. doi: 10.1074/jbc.M411925200. Epub 2004 Nov 16. J Biol Chem. 2005. PMID: 15546867 Free article.
We have recently shown that induction of biglycan (BGN) expression by transforming growth factor-beta1 (TGF-beta1) required sequential activation of both Smad and p38 mitogen-activated protein kinase signaling (Ungefroren, H., Lenschow, W., Chen, W.-B., and Kalthoff …
We have recently shown that induction of biglycan (BGN) expression by transforming growth factor-beta1 (TGF-beta1) required sequential activ …
Differentiation of in vitro-modified human peripheral blood monocytes into hepatocyte-like and pancreatic islet-like cells.
Ruhnke M, Ungefroren H, Nussler A, Martin F, Brulport M, Schormann W, Hengstler JG, Klapper W, Ulrichs K, Hutchinson JA, Soria B, Parwaresch RM, Heeckt P, Kremer B, Fändrich F. Ruhnke M, et al. Among authors: ungefroren h. Gastroenterology. 2005 Jun;128(7):1774-86. doi: 10.1053/j.gastro.2005.03.029. Gastroenterology. 2005. PMID: 15940611
146 results