Celsi G - Search Results

1

Prenatal exposure to high level of glucocorticoids increases the susceptibility of renal proximal tubular cells to apoptosis induced by uropathogenic Escherichia coli toxins.

Chen M, Bao W, Ceccatelli S, Celsi G. Chen M, et al. Among authors: celsi g. Am J Nephrol. 2004 Sep-Oct;24(5):497-502. doi: 10.1159/000080726. Epub 2004 Sep 8. Am J Nephrol. 2004. PMID: 15353912

2

Uropathogenic Escherichia coli toxins induce caspase-independent apoptosis in renal proximal tubular cells via ERK signaling.

Chen M, Jahnukainen T, Bao W, Daré E, Ceccatelli S, Celsi G. Chen M, et al. Among authors: celsi g. Am J Nephrol. 2003 May-Jun;23(3):140-51. doi: 10.1159/000069853. Epub 2003 Mar 6. Am J Nephrol. 2003. PMID: 12624487

3

Alterations in the intrauterine environment by glucocorticoids modifies the developmental programme of the auditory system.

Canlon B, Erichsen S, Nemlander E, Chen M, Hossain A, Celsi G, Ceccatelli S. Canlon B, et al. Among authors: celsi g. Eur J Neurosci. 2003 May;17(10):2035-41. doi: 10.1046/j.1460-9568.2003.02641.x. Eur J Neurosci. 2003. PMID: 12786969

4

Activation of extracellular signal-regulated kinase mediates apoptosis induced by uropathogenic Escherichia coli toxins via nitric oxide synthase: protective role of heme oxygenase-1.

Chen M, Bao W, Aizman R, Huang P, Aspevall O, Gustafsson LE, Ceccatelli S, Celsi G. Chen M, et al. Among authors: celsi g. J Infect Dis. 2004 Jul 1;190(1):127-35. doi: 10.1086/421243. Epub 2004 Jun 2. J Infect Dis. 2004. PMID: 15195252

5

Carbon monoxide prevents apoptosis induced by uropathogenic Escherichia coli toxins.

Chen M, Tofighi R, Bao W, Aspevall O, Jahnukainen T, Gustafsson LE, Ceccatelli S, Celsi G. Chen M, et al. Among authors: celsi g. Pediatr Nephrol. 2006 Mar;21(3):382-9. doi: 10.1007/s00467-005-2140-1. Epub 2005 Dec 31. Pediatr Nephrol. 2006. PMID: 16388391

6

N-acetylcysteine amide protects renal proximal tubular epithelial cells against iohexol-induced apoptosis by blocking p38 MAPK and iNOS signaling.

Gong X, Celsi G, Carlsson K, Norgren S, Chen M. Gong X, et al. Among authors: celsi g. Am J Nephrol. 2010;31(2):178-88. doi: 10.1159/000268161. Epub 2009 Dec 17. Am J Nephrol. 2010. PMID: 20016144

7

Prenatal exposure to high levels of glucocorticoids increases the susceptibility of cerebellar granule cells to oxidative stress-induced cell death.

Ahlbom E, Gogvadze V, Chen M, Celsi G, Ceccatelli S. Ahlbom E, et al. Among authors: celsi g. Proc Natl Acad Sci U S A. 2000 Dec 19;97(26):14726-30. doi: 10.1073/pnas.260501697. Proc Natl Acad Sci U S A. 2000. PMID: 11114198 Free PMC article.

8

Mechanisms of renal damage owing to infection.

Jahnukainen T, Chen M, Celsi G. Jahnukainen T, et al. Among authors: celsi g. Pediatr Nephrol. 2005 Aug;20(8):1043-53. doi: 10.1007/s00467-005-1898-5. Epub 2005 May 12. Pediatr Nephrol. 2005. PMID: 15889280 Review.

9

Prenatal dexamethasone causes oligonephronia, sodium retention, and higher blood pressure in the offspring.

Celsi G, Kistner A, Aizman R, Eklöf AC, Ceccatelli S, de Santiago A, Jacobson SH. Celsi G, et al. Pediatr Res. 1998 Sep;44(3):317-22. doi: 10.1203/00006450-199809000-00009. Pediatr Res. 1998. PMID: 9727707

10

Antenatal glucocorticoids and renal function after birth.

Jahnukainen T, Chen M, Berg U, Celsi G. Jahnukainen T, et al. Among authors: celsi g. Semin Neonatol. 2001 Aug;6(4):351-5. doi: 10.1053/siny.2001.0070. Semin Neonatol. 2001. PMID: 11972436 Review.

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