Koide H - Search Results

1

Kohno T, Koide H, Yokoyama S, Miyazawa T. Kohno T, et al. Among authors: koide h. Tanpakushitsu Kakusan Koso. 1992 Feb;37(3 Suppl):646-53. Tanpakushitsu Kakusan Koso. 1992. PMID: 1549738 Review. Japanese. No abstract available.

2

[Biosynthesis of alloprotein].

Koide H, Yokoyama S, Miyazawa T. Koide H, et al. Nihon Rinsho. 1990 Jan;48(1):208-13. Nihon Rinsho. 1990. PMID: 2406480 Review. Japanese. No abstract available.

3

Biosynthesis of a protein containing a nonprotein amino acid by Escherichia coli: L-2-aminohexanoic acid at position 21 in human epidermal growth factor.

Koide H, Yokoyama S, Kawai G, Ha JM, Oka T, Kawai S, Miyake T, Fuwa T, Miyazawa T. Koide H, et al. Proc Natl Acad Sci U S A. 1988 Sep;85(17):6237-41. doi: 10.1073/pnas.85.17.6237. Proc Natl Acad Sci U S A. 1988. PMID: 3045813 Free PMC article.

4

Receptor-binding affinities of human epidermal growth factor variants having unnatural amino acid residues in position 23.

Koide H, Yokoyama S, Katayama Y, Muto Y, Kigawa T, Kohno T, Takusari H, Oishi M, Takahashi S, Tsukumo K, et al. Koide H, et al. Biochemistry. 1994 Jun 14;33(23):7470-6. doi: 10.1021/bi00189a055. Biochemistry. 1994. PMID: 8003512

5

Site-directed mutagenesis, fluorescence, and two-dimensional NMR studies on microenvironments of effector region aromatic residues of human c-Ha-Ras protein.

Yamasaki K, Shirouzu M, Muto Y, Fujita-Yoshigaki J, Koide H, Ito Y, Kawai G, Hattori S, Yokoyama S, Nishimura S, et al. Yamasaki K, et al. Among authors: koide h. Biochemistry. 1994 Jan 11;33(1):65-73. doi: 10.1021/bi00167a009. Biochemistry. 1994. PMID: 8286364

6

c-Ha-Ras mutants with point mutations in Gln-Val-Val region have reduced inhibitory activity toward cathepsin B.

Hiwasa T, Fujita-Yoshigaki J, Shirouzu M, Koide H, Sawada T, Sakiyama S, Yokoyama S. Hiwasa T, et al. Among authors: koide h. Cancer Lett. 1993 May 14;69(3):161-5. doi: 10.1016/0304-3835(93)90169-a. Cancer Lett. 1993. PMID: 8513441

7

A glutamic acid residue at position 31 of Ras protein is essential to the signal transduction for neurite outgrowth of PC12 cells and the stimulation of GTPase activity by GAPRas.

Shirouzu M, Fujita-Yoshigaki J, Ito Y, Koide H, Nishimura S, Yokoyama S. Shirouzu M, et al. Among authors: koide h. Oncogene. 1992 Mar;7(3):475-80. Oncogene. 1992. PMID: 1549361

8

Identification of amino acid residues of Ras protein that are essential for signal-transducing activity but not for enhancement of GTPase activity by GAP.

Fujita-Yoshigaki J, Shirouzu M, Koide H, Nishimura S, Yokoyama S. Fujita-Yoshigaki J, et al. Among authors: koide h. FEBS Lett. 1991 Dec 9;294(3):187-90. doi: 10.1016/0014-5793(91)80665-p. FEBS Lett. 1991. PMID: 1756860 Free article.

9

Mutations that abolish the ability of Ha-Ras to associate with Raf-1.

Shirouzu M, Koide H, Fujita-Yoshigaki J, Oshio H, Toyama Y, Yamasaki K, Fuhrman SA, Villafranca E, Kaziro Y, Yokoyama S. Shirouzu M, et al. Among authors: koide h. Oncogene. 1994 Aug;9(8):2153-7. Oncogene. 1994. PMID: 8036000

10

Anti-(Raf-1) RNA aptamers that inhibit Ras-induced Raf-1 activation.

Kimoto M, Shirouzu M, Mizutani S, Koide H, Kaziro Y, Hirao I, Yokoyama S. Kimoto M, et al. Among authors: koide h. Eur J Biochem. 2002 Jan;269(2):697-704. doi: 10.1046/j.0014-2956.2001.02703.x. Eur J Biochem. 2002. PMID: 11856330 Free article.

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