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Page 1
Clinical and neuropathologic variation in neuronal intermediate filament inclusion disease.
Cairns NJ, Grossman M, Arnold SE, Burn DJ, Jaros E, Perry RH, Duyckaerts C, Stankoff B, Pillon B, Skullerud K, Cruz-Sanchez FF, Bigio EH, Mackenzie IR, Gearing M, Juncos JL, Glass JD, Yokoo H, Nakazato Y, Mosaheb S, Thorpe JR, Uryu K, Lee VM, Trojanowski JQ. Cairns NJ, et al. Among authors: thorpe jr. Neurology. 2004 Oct 26;63(8):1376-84. doi: 10.1212/01.wnl.0000139809.16817.dd. Neurology. 2004. PMID: 15505152 Free PMC article.
FUS immunogold labeling TEM analysis of the neuronal cytoplasmic inclusions of neuronal intermediate filament inclusion disease: a frontotemporal lobar degeneration with FUS proteinopathy.
Page T, Gitcho MA, Mosaheb S, Carter D, Chakraverty S, Perry RH, Bigio EH, Gearing M, Ferrer I, Goate AM, Cairns NJ, Thorpe JR. Page T, et al. Among authors: thorpe jr. J Mol Neurosci. 2011 Nov;45(3):409-21. doi: 10.1007/s12031-011-9549-8. Epub 2011 May 21. J Mol Neurosci. 2011. PMID: 21603978 Free PMC article.
Overproduction of a conserved domain of fission yeast and mammalian translation initiation factor eIF4G causes aberrant cell morphology and results in disruption of the localization of F-actin and the organization of microtubules.
Hashemzadeh-Bonehi L, Curtis PS, Morley SJ, Thorpe JR, Pain VM. Hashemzadeh-Bonehi L, et al. Among authors: thorpe jr. Genes Cells. 2003 Feb;8(2):163-78. doi: 10.1046/j.1365-2443.2003.00623.x. Genes Cells. 2003. PMID: 12581158 Free article.
363 results