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Page 1
Zmpste24 deficiency in mice causes spontaneous bone fractures, muscle weakness, and a prelamin A processing defect.
Bergo MO, Gavino B, Ross J, Schmidt WK, Hong C, Kendall LV, Mohr A, Meta M, Genant H, Jiang Y, Wisner ER, Van Bruggen N, Carano RA, Michaelis S, Griffey SM, Young SG. Bergo MO, et al. Among authors: meta m. Proc Natl Acad Sci U S A. 2002 Oct 1;99(20):13049-54. doi: 10.1073/pnas.192460799. Epub 2002 Sep 16. Proc Natl Acad Sci U S A. 2002. PMID: 12235369 Free PMC article.
Prelamin A and lamin A appear to be dispensable in the nuclear lamina.
Fong LG, Ng JK, Lammerding J, Vickers TA, Meta M, Coté N, Gavino B, Qiao X, Chang SY, Young SR, Yang SH, Stewart CL, Lee RT, Bennett CF, Bergo MO, Young SG. Fong LG, et al. Among authors: meta m. J Clin Invest. 2006 Mar;116(3):743-52. doi: 10.1172/JCI27125. J Clin Invest. 2006. PMID: 16511604 Free PMC article.
Protein farnesyltransferase inhibitors and progeria.
Meta M, Yang SH, Bergo MO, Fong LG, Young SG. Meta M, et al. Trends Mol Med. 2006 Oct;12(10):480-7. doi: 10.1016/j.molmed.2006.08.006. Epub 2006 Aug 30. Trends Mol Med. 2006. PMID: 16942914 Review.
Prelamin A farnesylation and progeroid syndromes.
Young SG, Meta M, Yang SH, Fong LG. Young SG, et al. Among authors: meta m. J Biol Chem. 2006 Dec 29;281(52):39741-5. doi: 10.1074/jbc.R600033200. Epub 2006 Nov 7. J Biol Chem. 2006. PMID: 17090536 Free article. Review.
Functional coupling between the extracellular matrix and nuclear lamina by Wnt signaling in progeria.
Hernandez L, Roux KJ, Wong ES, Mounkes LC, Mutalif R, Navasankari R, Rai B, Cool S, Jeong JW, Wang H, Lee HS, Kozlov S, Grunert M, Keeble T, Jones CM, Meta MD, Young SG, Daar IO, Burke B, Perantoni AO, Stewart CL. Hernandez L, et al. Among authors: meta md. Dev Cell. 2010 Sep 14;19(3):413-25. doi: 10.1016/j.devcel.2010.08.013. Dev Cell. 2010. PMID: 20833363 Free PMC article.
53 results