Prowse AH - Search Results

1

Allelic loss studies do not provide evidence for the "endometriosis-as-tumor" theory.

Prowse AH, Fakis G, Manek S, Churchman M, Edwards S, Rowan A, Koninckx P, Kennedy S, Tomlinson IP. Prowse AH, et al. Fertil Steril. 2005 Apr;83 Suppl 1:1134-43. doi: 10.1016/j.fertnstert.2004.07.982. Fertil Steril. 2005. PMID: 15831286 Free article.

2

Molecular genetic evidence that endometriosis is a precursor of ovarian cancer.

Prowse AH, Manek S, Varma R, Liu J, Godwin AK, Maher ER, Tomlinson IP, Kennedy SH. Prowse AH, et al. Int J Cancer. 2006 Aug 1;119(3):556-62. doi: 10.1002/ijc.21845. Int J Cancer. 2006. PMID: 16506222

3

Inactivation of the von Hippel-Lindau (VHL) tumour suppressor gene and allelic losses at chromosome arm 3p in primary renal cell carcinoma: evidence for a VHL-independent pathway in clear cell renal tumourigenesis.

Clifford SC, Prowse AH, Affara NA, Buys CH, Maher ER. Clifford SC, et al. Among authors: prowse ah. Genes Chromosomes Cancer. 1998 Jul;22(3):200-9. doi: 10.1002/(sici)1098-2264(199807)22:3<200::aid-gcc5>3.0.co;2-#. Genes Chromosomes Cancer. 1998. PMID: 9624531

4

OVCA2 is downregulated and degraded during retinoid-induced apoptosis.

Prowse AH, Vanderveer L, Milling SW, Pan ZZ, Dunbrack RL, Xu XX, Godwin AK. Prowse AH, et al. Int J Cancer. 2002 May 10;99(2):185-92. doi: 10.1002/ijc.10334. Int J Cancer. 2002. PMID: 11979432

5

DNA array-based method for detection of large rearrangements in the BRCA1 gene.

Frolov A, Prowse AH, Vanderveer L, Bove B, Wu H, Godwin AK. Frolov A, et al. Among authors: prowse ah. Genes Chromosomes Cancer. 2002 Nov;35(3):232-41. doi: 10.1002/gcc.10109. Genes Chromosomes Cancer. 2002. PMID: 12353265

6

Expression of OVCA1, a candidate tumor suppressor, is reduced in tumors and inhibits growth of ovarian cancer cells.

Bruening W, Prowse AH, Schultz DC, Holgado-Madruga M, Wong A, Godwin AK. Bruening W, et al. Among authors: prowse ah. Cancer Res. 1999 Oct 1;59(19):4973-83. Cancer Res. 1999. PMID: 10519411

7

Identification of a splice acceptor site mutation in p16INK4A/p14ARF within a breast cancer, melanoma, neurofibroma prone kindred.

Prowse AH, Schultz DC, Guo S, Vanderveer L, Dangel J, Bove B, Cairns P, Daly M, Godwin AK. Prowse AH, et al. J Med Genet. 2003 Aug;40(8):e102. doi: 10.1136/jmg.40.8.e102. J Med Genet. 2003. PMID: 12920094 Free PMC article. No abstract available.

8

Disabled-2 mediates c-Fos suppression and the cell growth regulatory activity of retinoic acid in embryonic carcinoma cells.

Smith ER, Capo-chichi CD, He J, Smedberg JL, Yang DH, Prowse AH, Godwin AK, Hamilton TC, Xu XX. Smith ER, et al. Among authors: prowse ah. J Biol Chem. 2001 Dec 14;276(50):47303-10. doi: 10.1074/jbc.M106158200. Epub 2001 Sep 27. J Biol Chem. 2001. PMID: 11577091 Free article.

9

Somatic inactivation of the VHL gene in Von Hippel-Lindau disease tumors.

Prowse AH, Webster AR, Richards FM, Richard S, Olschwang S, Resche F, Affara NA, Maher ER. Prowse AH, et al. Am J Hum Genet. 1997 Apr;60(4):765-71. Am J Hum Genet. 1997. PMID: 9106522 Free PMC article.

10

Somatic mutations of the von Hippel-Lindau disease tumour suppressor gene in non-familial clear cell renal carcinoma.

Foster K, Prowse A, van den Berg A, Fleming S, Hulsbeek MM, Crossey PA, Richards FM, Cairns P, Affara NA, Ferguson-Smith MA, et al. Foster K, et al. Hum Mol Genet. 1994 Dec;3(12):2169-73. doi: 10.1093/hmg/3.12.2169. Hum Mol Genet. 1994. PMID: 7881415

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