Sho E - Search Results

1

Intermittent short-duration exposure to low wall shear stress induces intimal thickening in arteries exposed to chronic high shear stress.

Nanjo H, Sho E, Komatsu M, Sho M, Zarins CK, Masuda H. Nanjo H, et al. Among authors: sho m, sho e. Exp Mol Pathol. 2006 Feb;80(1):38-45. doi: 10.1016/j.yexmp.2005.04.005. Epub 2005 Jun 15. Exp Mol Pathol. 2006. PMID: 15961075

2

Subnormal shear stress-induced intimal thickening requires medial smooth muscle cell proliferation and migration.

Sho M, Sho E, Singh TM, Komatsu M, Sugita A, Xu C, Nanjo H, Zarins CK, Masuda H. Sho M, et al. Among authors: sho e. Exp Mol Pathol. 2002 Apr;72(2):150-60. doi: 10.1006/exmp.2002.2426. Exp Mol Pathol. 2002. PMID: 11890724

3

Arterial enlargement in response to high flow requires early expression of matrix metalloproteinases to degrade extracellular matrix.

Sho E, Sho M, Singh TM, Nanjo H, Komatsu M, Xu C, Masuda H, Zarins CK. Sho E, et al. Among authors: sho m. Exp Mol Pathol. 2002 Oct;73(2):142-53. doi: 10.1006/exmp.2002.2457. Exp Mol Pathol. 2002. PMID: 12231217

4

Ultrastructure of endothelial cells under flow alteration.

Masuda H, Kawamura K, Nanjo H, Sho E, Komatsu M, Sugiyama T, Sugita A, Asari Y, Kobayashi M, Ebina T, Hoshi N, Singh TM, Xu C, Zarins CK. Masuda H, et al. Among authors: sho e. Microsc Res Tech. 2003 Jan 1;60(1):2-12. doi: 10.1002/jemt.10237. Microsc Res Tech. 2003. PMID: 12500255 Review.

5

Arterial enlargement, tortuosity, and intimal thickening in response to sequential exposure to high and low wall shear stress.

Sho E, Nanjo H, Sho M, Kobayashi M, Komatsu M, Kawamura K, Xu C, Zarins CK, Masuda H. Sho E, et al. Among authors: sho m. J Vasc Surg. 2004 Mar;39(3):601-12. doi: 10.1016/j.jvs.2003.10.058. J Vasc Surg. 2004. PMID: 14981455 Free article.

6

Normalization of high-flow or removal of flow cannot stop high-flow induced endothelial proliferation.

Yamauchi M, Takahashi M, Kobayashi M, Sho E, Nanjo H, Kawamura K, Masuda H. Yamauchi M, et al. Among authors: sho e. Biomed Res. 2005 Feb;26(1):21-8. doi: 10.2220/biomedres.26.21. Biomed Res. 2005. PMID: 15806980 Free article.

7

Blood flow decrease induces apoptosis of endothelial cells in previously dilated arteries resulting from chronic high blood flow.

Sho E, Sho M, Singh TM, Xu C, Zarins CK, Masuda H. Sho E, et al. Among authors: sho m. Arterioscler Thromb Vasc Biol. 2001 Jul;21(7):1139-45. doi: 10.1161/hq0701.092118. Arterioscler Thromb Vasc Biol. 2001. PMID: 11451742

8

High flow drives vascular endothelial cell proliferation during flow-induced arterial remodeling associated with the expression of vascular endothelial growth factor.

Sho E, Komatsu M, Sho M, Nanjo H, Singh TM, Xu C, Masuda H, Zarins CK. Sho E, et al. Among authors: sho m. Exp Mol Pathol. 2003 Aug;75(1):1-11. doi: 10.1016/s0014-4800(03)00032-7. Exp Mol Pathol. 2003. PMID: 12834620

9

Weaving hypothesis of cardiomyocyte sarcomeres: discovery of periodic broadening and narrowing of intercalated disk during volume-load change.

Yoshida M, Sho E, Nanjo H, Takahashi M, Kobayashi M, Kawamura K, Honma M, Komatsu M, Sugita A, Yamauchi M, Hosoi T, Ito Y, Masuda H. Yoshida M, et al. Among authors: sho e. Am J Pathol. 2010 Feb;176(2):660-78. doi: 10.2353/ajpath.2010.090348. Epub 2010 Jan 7. Am J Pathol. 2010. PMID: 20056839 Free PMC article.

10

Wall shear stress and strain modulate experimental aneurysm cellularity.

Hoshina K, Sho E, Sho M, Nakahashi TK, Dalman RL. Hoshina K, et al. Among authors: sho m, sho e. J Vasc Surg. 2003 May;37(5):1067-74. doi: 10.1016/s0741-5214(03)70052-4. J Vasc Surg. 2003. PMID: 12756356 Free article.

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