Launois P - Search Results

1

LACK-reactive CD4+ T cells require autocrine IL-2 to mediate susceptibility to Leishmania major.

Gumy A, Aseffa A, Rachinel N, Breton M, Otten L, Tacchini-Cottier F, Röcken M, Doyen N, Acha-Orbea H, Locksley RM, MacDonald HR, Launois P, Louis J. Gumy A, et al. Among authors: launois p. Eur J Immunol. 2006 Jun;36(6):1465-73. doi: 10.1002/eji.200535801. Eur J Immunol. 2006. PMID: 16637008 Free article.

2

Different epitopes of the LACK protein are recognized by V beta 4 V alpha 8 CD4+ T cells in H-2b and H-2d mice susceptible to Leishmania major.

Launois P, Pingel S, Himmelrich H, Locksley R, Louis J. Launois P, et al. Microbes Infect. 2007 Sep;9(11):1260-6. doi: 10.1016/j.micinf.2007.05.017. Epub 2007 May 27. Microbes Infect. 2007. PMID: 17890126 Free article.

3

An essential role for transmembrane TNF in the resolution of the inflammatory lesion induced by Leishmania major infection.

Allenbach C, Launois P, Mueller C, Tacchini-Cottier F. Allenbach C, et al. Among authors: launois p. Eur J Immunol. 2008 Mar;38(3):720-31. doi: 10.1002/eji.200737662. Eur J Immunol. 2008. PMID: 18266271 Free article.

4

Leishmania major-specific B cells are necessary for Th2 cell development and susceptibility to L. major LV39 in BALB/c mice.

Ronet C, Voigt H, Himmelrich H, Doucey MA, Hauyon-La Torre Y, Revaz-Breton M, Tacchini-Cottier F, Bron C, Louis J, Launois P. Ronet C, et al. Among authors: launois p. J Immunol. 2008 Apr 1;180(7):4825-35. doi: 10.4049/jimmunol.180.7.4825. J Immunol. 2008. PMID: 18354206 Free article.

5

Regulatory B cells shape the development of Th2 immune responses in BALB/c mice infected with Leishmania major through IL-10 production.

Ronet C, Hauyon-La Torre Y, Revaz-Breton M, Mastelic B, Tacchini-Cottier F, Louis J, Launois P. Ronet C, et al. Among authors: launois p. J Immunol. 2010 Jan 15;184(2):886-94. doi: 10.4049/jimmunol.0901114. Epub 2009 Dec 4. J Immunol. 2010. PMID: 19966209

6

The MyD88 protein 88 pathway is differently involved in immune responses induced by distinct substrains of Leishmania major.

Revaz-Breton M, Ronet C, Ives A, Torre YH, Masina S, Tacchini-Cottier F, Launois P. Revaz-Breton M, et al. Among authors: launois p. Eur J Immunol. 2010 Jun;40(6):1697-707. doi: 10.1002/eji.200939821. Eur J Immunol. 2010. PMID: 20333623 Free article.

7

Rapid IL-4 production by Leishmania homolog of mammalian RACK1-reactive CD4(+) T cells in resistant mice treated once with anti-IL-12 or -IFN-gamma antibodies at the onset of infection with Leishmania major instructs Th2 cell development, resulting in nonhealing lesions.

Launois P, Gumy A, Himmelrich H, Locksley RM, Röcken M, Louis JA. Launois P, et al. J Immunol. 2002 May 1;168(9):4628-35. doi: 10.4049/jimmunol.168.9.4628. J Immunol. 2002. PMID: 11971011

8

Experimental cutaneous Leishmaniasis: a powerful model to study in vivo the mechanisms underlying genetic differences in Th subset differentiation.

Louis J, Gumy A, Voigt H, Röcken M, Launois P. Louis J, et al. Among authors: launois p. Eur J Dermatol. 2002 Jul-Aug;12(4):316-8. Eur J Dermatol. 2002. PMID: 12095873 Review.

9

The early IL-4 response to Leishmania major and the resulting Th2 cell maturation steering progressive disease in BALB/c mice are subject to the control of regulatory CD4+CD25+ T cells.

Aseffa A, Gumy A, Launois P, MacDonald HR, Louis JA, Tacchini-Cottier F. Aseffa A, et al. Among authors: launois p. J Immunol. 2002 Sep 15;169(6):3232-41. doi: 10.4049/jimmunol.169.6.3232. J Immunol. 2002. PMID: 12218142

10

The murine model of infection with Leishmania major and its importance for the deciphering of mechanisms underlying differences in Th cell differentiation in mice from different genetic backgrounds.

Gumy A, Louis JA, Launois P. Gumy A, et al. Among authors: launois p. Int J Parasitol. 2004 Mar 29;34(4):433-44. doi: 10.1016/j.ijpara.2003.11.021. Int J Parasitol. 2004. PMID: 15013733 Review.

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