Su YD - Search Results

1

Inhibition of mTOR reduces chronic pressure-overload cardiac hypertrophy and fibrosis.

Gao XM, Wong G, Wang B, Kiriazis H, Moore XL, Su YD, Dart A, Du XJ. Gao XM, et al. Among authors: su yd. J Hypertens. 2006 Aug;24(8):1663-70. doi: 10.1097/01.hjh.0000239304.01496.83. J Hypertens. 2006. PMID: 16877971

2

Knockout of beta(1)- and beta(2)-adrenoceptors attenuates pressure overload-induced cardiac hypertrophy and fibrosis.

Kiriazis H, Wang K, Xu Q, Gao XM, Ming Z, Su Y, Moore XL, Lambert G, Gibbs ME, Dart AM, Du XJ. Kiriazis H, et al. Br J Pharmacol. 2008 Feb;153(4):684-92. doi: 10.1038/sj.bjp.0707622. Epub 2008 Jan 14. Br J Pharmacol. 2008. PMID: 18193078 Free PMC article.

3

Relaxin reverses cardiac and renal fibrosis in spontaneously hypertensive rats.

Lekgabe ED, Kiriazis H, Zhao C, Xu Q, Moore XL, Su Y, Bathgate RA, Du XJ, Samuel CS. Lekgabe ED, et al. Hypertension. 2005 Aug;46(2):412-8. doi: 10.1161/01.HYP.0000171930.00697.2f. Epub 2005 Jun 20. Hypertension. 2005. PMID: 15967869

4

Chronic activation of the low affinity site of β1-adrenoceptors stimulates haemodynamics but exacerbates pressure-overload cardiac remodelling.

Kiriazis H, Tugiono N, Xu Q, Gao XM, Jennings NL, Ming Z, Su Y, Klenowski P, Summers RJ, Kaumann A, Molenaar P, Du XJ. Kiriazis H, et al. Br J Pharmacol. 2013 Sep;170(2):352-65. doi: 10.1111/bph.12272. Br J Pharmacol. 2013. PMID: 23750586 Free PMC article.

5

Inhibition of miR-154 Protects Against Cardiac Dysfunction and Fibrosis in a Mouse Model of Pressure Overload.

Bernardo BC, Nguyen SS, Gao XM, Tham YK, Ooi JY, Patterson NL, Kiriazis H, Su Y, Thomas CJ, Lin RC, Du XJ, McMullen JR. Bernardo BC, et al. Sci Rep. 2016 Mar 1;6:22442. doi: 10.1038/srep22442. Sci Rep. 2016. PMID: 26928825 Free PMC article.

6

Relaxin antagonizes hypertrophy and apoptosis in neonatal rat cardiomyocytes.

Moore XL, Tan SL, Lo CY, Fang L, Su YD, Gao XM, Woodcock EA, Summers RJ, Tregear GW, Bathgate RA, Du XJ. Moore XL, et al. Among authors: su yd. Endocrinology. 2007 Apr;148(4):1582-9. doi: 10.1210/en.2006-1324. Epub 2007 Jan 4. Endocrinology. 2007. PMID: 17204550

7

Transgenic alpha1A-adrenergic activation limits post-infarct ventricular remodeling and dysfunction and improves survival.

Du XJ, Gao XM, Kiriazis H, Moore XL, Ming Z, Su Y, Finch AM, Hannan RA, Dart AM, Graham RM. Du XJ, et al. Cardiovasc Res. 2006 Sep 1;71(4):735-43. doi: 10.1016/j.cardiores.2006.06.015. Epub 2006 Jun 16. Cardiovasc Res. 2006. PMID: 16859660

8

Differences in inflammation, MMP activation and collagen damage account for gender difference in murine cardiac rupture following myocardial infarction.

Fang L, Gao XM, Moore XL, Kiriazis H, Su Y, Ming Z, Lim YL, Dart AM, Du XJ. Fang L, et al. J Mol Cell Cardiol. 2007 Nov;43(5):535-44. doi: 10.1016/j.yjmcc.2007.06.011. Epub 2007 Jul 10. J Mol Cell Cardiol. 2007. PMID: 17689559

9

Higher levels of collagen and facilitated healing protect against ventricular rupture following myocardial infarction.

Fang L, Gao XM, Samuel CS, Su Y, Lim YL, Dart AM, Du XJ. Fang L, et al. Clin Sci (Lond). 2008 Aug;115(3):99-106. doi: 10.1042/CS20070365. Clin Sci (Lond). 2008. PMID: 18248324

10

Infarct size and post-infarct inflammation determine the risk of cardiac rupture in mice.

Gao XM, Ming Z, Su Y, Fang L, Kiriazis H, Xu Q, Dart AM, Du XJ. Gao XM, et al. Int J Cardiol. 2010 Aug 6;143(1):20-8. doi: 10.1016/j.ijcard.2009.01.019. Epub 2009 Feb 4. Int J Cardiol. 2010. PMID: 19195725

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