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Repopulation of apolipoprotein E knockout mice with CCR2-deficient bone marrow progenitor cells does not inhibit ongoing atherosclerotic lesion development.
Guo J, de Waard V, Van Eck M, Hildebrand RB, van Wanrooij EJ, Kuiper J, Maeda N, Benson GM, Groot PH, Van Berkel TJ. Guo J, et al. Among authors: hildebrand rb. Arterioscler Thromb Vasc Biol. 2005 May;25(5):1014-9. doi: 10.1161/01.ATV.0000163181.40896.42. Epub 2005 Mar 17. Arterioscler Thromb Vasc Biol. 2005. PMID: 15774908
Macrophage ABCG1 deletion disrupts lipid homeostasis in alveolar macrophages and moderately influences atherosclerotic lesion development in LDL receptor-deficient mice.
Out R, Hoekstra M, Hildebrand RB, Kruit JK, Meurs I, Li Z, Kuipers F, Van Berkel TJ, Van Eck M. Out R, et al. Among authors: hildebrand rb. Arterioscler Thromb Vasc Biol. 2006 Oct;26(10):2295-300. doi: 10.1161/01.ATV.0000237629.29842.4c. Epub 2006 Jul 20. Arterioscler Thromb Vasc Biol. 2006. PMID: 16857950
Important role for bone marrow-derived cholesteryl ester transfer protein in lipoprotein cholesterol redistribution and atherosclerotic lesion development in LDL receptor knockout mice.
Van Eck M, Ye D, Hildebrand RB, Kar Kruijt J, de Haan W, Hoekstra M, Rensen PC, Ehnholm C, Jauhiainen M, Van Berkel TJ. Van Eck M, et al. Among authors: hildebrand rb. Circ Res. 2007 Mar 16;100(5):678-85. doi: 10.1161/01.RES.0000260202.79927.4f. Epub 2007 Feb 9. Circ Res. 2007. PMID: 17293475
Combined deletion of macrophage ABCA1 and ABCG1 leads to massive lipid accumulation in tissue macrophages and distinct atherosclerosis at relatively low plasma cholesterol levels.
Out R, Hoekstra M, Habets K, Meurs I, de Waard V, Hildebrand RB, Wang Y, Chimini G, Kuiper J, Van Berkel TJ, Van Eck M. Out R, et al. Among authors: hildebrand rb. Arterioscler Thromb Vasc Biol. 2008 Feb;28(2):258-64. doi: 10.1161/ATVBAHA.107.156935. Epub 2007 Nov 15. Arterioscler Thromb Vasc Biol. 2008. PMID: 18006857
37 results