Mondragón-Rodríguez S - Search Results

1

Truncation of tau protein and its pathological significance in Alzheimer's disease.

García-Sierra F, Mondragón-Rodríguez S, Basurto-Islas G. García-Sierra F, et al. J Alzheimers Dis. 2008 Aug;14(4):401-9. doi: 10.3233/jad-2008-14407. J Alzheimers Dis. 2008. PMID: 18688090 Review.

2

Conformational changes and cleavage of tau in Pick bodies parallel the early processing of tau found in Alzheimer pathology.

Mondragón-Rodríguez S, Mena R, Binder LI, Smith MA, Perry G, García-Sierra F. Mondragón-Rodríguez S, et al. Neuropathol Appl Neurobiol. 2008 Feb;34(1):62-75. doi: 10.1111/j.1365-2990.2007.00853.x. Epub 2007 Oct 26. Neuropathol Appl Neurobiol. 2008. PMID: 17971079

3

Cleavage and conformational changes of tau protein follow phosphorylation during Alzheimer's disease.

Mondragón-Rodríguez S, Basurto-Islas G, Santa-Maria I, Mena R, Binder LI, Avila J, Smith MA, Perry G, García-Sierra F. Mondragón-Rodríguez S, et al. Int J Exp Pathol. 2008 Apr;89(2):81-90. doi: 10.1111/j.1365-2613.2007.00568.x. Int J Exp Pathol. 2008. PMID: 18336525 Free PMC article.

4

Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

Mondragón-Rodríguez S, Basurto-Islas G, Lee HG, Perry G, Zhu X, Castellani RJ, Smith MA. Mondragón-Rodríguez S, et al. Expert Rev Neurother. 2010 May;10(5):683-91. doi: 10.1586/ern.10.27. Expert Rev Neurother. 2010. PMID: 20420489 Free PMC article. Review.

5

Phosphorylation of tau protein as the link between oxidative stress, mitochondrial dysfunction, and connectivity failure: implications for Alzheimer's disease.

Mondragón-Rodríguez S, Perry G, Zhu X, Moreira PI, Acevedo-Aquino MC, Williams S. Mondragón-Rodríguez S, et al. Oxid Med Cell Longev. 2013;2013:940603. doi: 10.1155/2013/940603. Epub 2013 Jul 10. Oxid Med Cell Longev. 2013. PMID: 23936615 Free PMC article. Review.

6

Tau, Amyloid Beta and Deep Brain Stimulation: Aiming to Restore Cognitive Deficit in Alzheimer's Disease.

Mondragón-Rodríguez S, Perry G, Pena-Ortega F, Williams S. Mondragón-Rodríguez S, et al. Curr Alzheimer Res. 2017;14(1):40-46. doi: 10.2174/1567205013666160819131336. Curr Alzheimer Res. 2017. PMID: 27539594 Review.

7

Hyperphosphorylated Tau Relates to Improved Cognitive Performance and Reduced Hippocampal Excitability in the Young rTg4510 Mouse Model of Tauopathy.

Xolalpa-Cueva L, García-Carlos CA, Villaseñor-Zepeda R, Orta-Salazar E, Díaz-Cintra S, Peña-Ortega F, Perry G, Mondragón-Rodríguez S. Xolalpa-Cueva L, et al. Among authors: mondragon rodriguez s. J Alzheimers Dis. 2022;87(2):529-543. doi: 10.3233/JAD-215186. J Alzheimers Dis. 2022. PMID: 35342085

8

Meta-Analysis in Transgenic Alzheimer's Disease Mouse Models Reveals Opposite Brain Network Effects of Amyloid-β and Phosphorylated Tau Proteins.

García-Carlos CA, Basurto-Islas G, Perry G, Mondragón-Rodríguez S. García-Carlos CA, et al. Among authors: mondragon rodriguez s. J Alzheimers Dis. 2024;99(2):595-607. doi: 10.3233/JAD-231365. J Alzheimers Dis. 2024. PMID: 38669540

9

Amyloid Beta and tau proteins as therapeutic targets for Alzheimer's disease treatment: rethinking the current strategy.

Mondragón-Rodríguez S, Perry G, Zhu X, Boehm J. Mondragón-Rodríguez S, et al. Int J Alzheimers Dis. 2012;2012:630182. doi: 10.1155/2012/630182. Epub 2012 Mar 8. Int J Alzheimers Dis. 2012. PMID: 22482074 Free PMC article.

10

Phosphorylation of tau protein at sites Ser(396-404) is one of the earliest events in Alzheimer's disease and Down syndrome.

Mondragón-Rodríguez S, Perry G, Luna-Muñoz J, Acevedo-Aquino MC, Williams S. Mondragón-Rodríguez S, et al. Neuropathol Appl Neurobiol. 2014 Feb;40(2):121-35. doi: 10.1111/nan.12084. Neuropathol Appl Neurobiol. 2014. PMID: 24033439

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