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p53: a cell cycle regulator activated by DNA damage.
Selivanova G, Wiman KG. Selivanova G, et al. Adv Cancer Res. 1995;66:143-80. doi: 10.1016/s0065-230x(08)60253-5. Adv Cancer Res. 1995. PMID: 7793313 Review. No abstract available.
Reactivation of mutant p53 through interaction of a C-terminal peptide with the core domain.
Selivanova G, Ryabchenko L, Jansson E, Iotsova V, Wiman KG. Selivanova G, et al. Mol Cell Biol. 1999 May;19(5):3395-402. doi: 10.1128/MCB.19.5.3395. Mol Cell Biol. 1999. PMID: 10207063 Free PMC article.
A synthetic 22-mer peptide (peptide 46) derived from the p53 C-terminal domain can restore the growth suppressor function of mutant p53 proteins in human tumor cells (G. Selivanova et al., Nat. Med. 3:632-638, 1997). Here we demonstrate that peptide 46 binds mutant …
A synthetic 22-mer peptide (peptide 46) derived from the p53 C-terminal domain can restore the growth suppressor function of mutant p53 prot …
p53-induced apoptosis as a safeguard against cancer.
Asker C, Wiman KG, Selivanova G. Asker C, et al. Among authors: selivanova g. Biochem Biophys Res Commun. 1999 Nov;265(1):1-6. doi: 10.1006/bbrc.1999.1446. Biochem Biophys Res Commun. 1999. PMID: 10548481 Review.
159 results