Van Dyke T - Search Results

1

Inactivation of SNF5 cooperates with p53 loss to accelerate tumor formation in Snf5(+/-);p53(+/-) mice.

DelBove J, Kuwahara Y, Mora-Blanco EL, Godfrey V, Funkhouser WK, Fletcher CD, Van Dyke T, Roberts CW, Weissman BE. DelBove J, et al. Among authors: van dyke t. Mol Carcinog. 2009 Dec;48(12):1139-48. doi: 10.1002/mc.20568. Mol Carcinog. 2009. PMID: 19676100 Free PMC article.

2

Tumor-specific cooperation of retinoblastoma protein family and Snf5 inactivation.

Chai J, Lu X, Godfrey V, Fletcher C, Roberts CW, Van Dyke T, Weissman BE. Chai J, et al. Among authors: van dyke t. Cancer Res. 2007 Apr 1;67(7):3002-9. doi: 10.1158/0008-5472.CAN-06-4207. Cancer Res. 2007. PMID: 17409406

3

The BRG1 chromatin remodeler protects against ovarian cysts, uterine tumors, and mammary tumors in a lineage-specific manner.

Serber DW, Rogala A, Makarem M, Rosson GB, Simin K, Godfrey V, Van Dyke T, Eaves CJ, Bultman SJ. Serber DW, et al. Among authors: van dyke t. PLoS One. 2012;7(2):e31346. doi: 10.1371/journal.pone.0031346. Epub 2012 Feb 21. PLoS One. 2012. PMID: 22363625 Free PMC article.

4

Selective inactivation of p53 facilitates mouse epithelial tumor progression without chromosomal instability.

Lu X, Magrane G, Yin C, Louis DN, Gray J, Van Dyke T. Lu X, et al. Among authors: van dyke t. Mol Cell Biol. 2001 Sep;21(17):6017-30. doi: 10.1128/MCB.21.17.6017-6030.2001. Mol Cell Biol. 2001. PMID: 11486039 Free PMC article.

5

Apoptosis is the essential target of selective pressure against p53, whereas loss of additional p53 functions facilitates carcinoma progression.

Lu X, Yang C, Yin C, Van Dyke T, Simin K. Lu X, et al. Among authors: van dyke t. Mol Cancer Res. 2011 Apr;9(4):430-9. doi: 10.1158/1541-7786.MCR-10-0277. Epub 2011 Mar 8. Mol Cancer Res. 2011. PMID: 21385880 Free PMC article.

6

Dietary antioxidant depletion: enhancement of tumor apoptosis and inhibition of brain tumor growth in transgenic mice.

Salganik RI, Albright CD, Rodgers J, Kim J, Zeisel SH, Sivashinskiy MS, Van Dyke TA. Salganik RI, et al. Among authors: van dyke ta. Carcinogenesis. 2000 May;21(5):909-14. doi: 10.1093/carcin/21.5.909. Carcinogenesis. 2000. PMID: 10783311

7

p19(ARF) is dispensable for oncogenic stress-induced p53-mediated apoptosis and tumor suppression in vivo.

Tolbert D, Lu X, Yin C, Tantama M, Van Dyke T. Tolbert D, et al. Among authors: van dyke t. Mol Cell Biol. 2002 Jan;22(1):370-7. doi: 10.1128/MCB.22.1.370-377.2002. Mol Cell Biol. 2002. PMID: 11739748 Free PMC article.

8

Key roles for E2F1 in signaling p53-dependent apoptosis and in cell division within developing tumors.

Pan H, Yin C, Dyson NJ, Harlow E, Yamasaki L, Van Dyke T. Pan H, et al. Among authors: van dyke t. Mol Cell. 1998 Sep;2(3):283-92. doi: 10.1016/s1097-2765(00)80273-7. Mol Cell. 1998. PMID: 9774967 Free article.

9

Tissue-specific inactivation of p53 tumor suppression in the mouse.

Bowman T, Symonds H, Gu L, Yin C, Oren M, Van Dyke T. Bowman T, et al. Among authors: van dyke t. Genes Dev. 1996 Apr 1;10(7):826-35. doi: 10.1101/gad.10.7.826. Genes Dev. 1996. PMID: 8846919 Free article.

10

Atm is dispensable for p53 apoptosis and tumor suppression triggered by cell cycle dysfunction.

Liao MJ, Yin C, Barlow C, Wynshaw-Boris A, van Dyke T. Liao MJ, et al. Among authors: van dyke t. Mol Cell Biol. 1999 Apr;19(4):3095-102. doi: 10.1128/MCB.19.4.3095. Mol Cell Biol. 1999. PMID: 10082576 Free PMC article.

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