Collingwood J - Search Results

1

Copper abolishes the beta-sheet secondary structure of preformed amyloid fibrils of amyloid-beta(42).

House E, Mold M, Collingwood J, Baldwin A, Goodwin S, Exley C. House E, et al. Among authors: collingwood j. J Alzheimers Dis. 2009;18(4):811-7. doi: 10.3233/JAD-2009-1235. J Alzheimers Dis. 2009. PMID: 19749401 Free PMC article.

2

Aluminium, iron, zinc and copper influence the in vitro formation of amyloid fibrils of Abeta42 in a manner which may have consequences for metal chelation therapy in Alzheimer's disease.

House E, Collingwood J, Khan A, Korchazkina O, Berthon G, Exley C. House E, et al. Among authors: collingwood j. J Alzheimers Dis. 2004 Jun;6(3):291-301. doi: 10.3233/jad-2004-6310. J Alzheimers Dis. 2004. PMID: 15201484

3

Mapping and characterization of iron compounds in Alzheimer's tissue.

Collingwood J, Dobson J. Collingwood J, et al. J Alzheimers Dis. 2006 Nov;10(2-3):215-22. doi: 10.3233/jad-2006-102-308. J Alzheimers Dis. 2006. PMID: 17119289 Review.

4

Three-dimensional tomographic imaging and characterization of iron compounds within Alzheimer's plaque core material.

Collingwood JF, Chong RK, Kasama T, Cervera-Gontard L, Dunin-Borkowski RE, Perry G, Pósfai M, Siedlak SL, Simpson ET, Smith MA, Dobson J. Collingwood JF, et al. J Alzheimers Dis. 2008 Jun;14(2):235-45. doi: 10.3233/jad-2008-14211. J Alzheimers Dis. 2008. PMID: 18560134

5

Spherulites of amyloid-beta42 in vitro and in Alzheimer's disease.

Exley C, House E, Collingwood JF, Davidson MR, Cannon D, Donald AM. Exley C, et al. J Alzheimers Dis. 2010;20(4):1159-65. doi: 10.3233/JAD-2010-091630. J Alzheimers Dis. 2010. PMID: 20413877 Free PMC article.

6

Iron deficiency in parkinsonism: region-specific iron dysregulation in Parkinson's disease and multiple system atrophy.

Visanji NP, Collingwood JF, Finnegan ME, Tandon A, House E, Hazrati LN. Visanji NP, et al. J Parkinsons Dis. 2013;3(4):523-37. doi: 10.3233/JPD-130197. J Parkinsons Dis. 2013. PMID: 24113558

7

Ferrous iron formation following the co-aggregation of ferric iron and the Alzheimer's disease peptide β-amyloid (1-42).

Everett J, Céspedes E, Shelford LR, Exley C, Collingwood JF, Dobson J, van der Laan G, Jenkins CA, Arenholz E, Telling ND. Everett J, et al. Among authors: collingwood jf. J R Soc Interface. 2014 Mar 26;11(95):20140165. doi: 10.1098/rsif.2014.0165. Print 2014 Jun 6. J R Soc Interface. 2014. PMID: 24671940 Free PMC article.

8

Synchrotron XRF imaging of Alzheimer's disease basal ganglia reveals linear dependence of high-field magnetic resonance microscopy on tissue iron concentration.

Finnegan ME, Visanji NP, Romero-Canelon I, House E, Rajan S, Mosselmans JFW, Hazrati LN, Dobson J, Collingwood JF. Finnegan ME, et al. Among authors: collingwood jf. J Neurosci Methods. 2019 May 1;319:28-39. doi: 10.1016/j.jneumeth.2019.03.002. Epub 2019 Mar 6. J Neurosci Methods. 2019. PMID: 30851339 Free article.

9

In situ characterization and mapping of iron compounds in Alzheimer's disease tissue.

Collingwood JF, Mikhaylova A, Davidson M, Batich C, Streit WJ, Terry J, Dobson J. Collingwood JF, et al. J Alzheimers Dis. 2005 Aug;7(4):267-72. doi: 10.3233/jad-2005-7401. J Alzheimers Dis. 2005. PMID: 16131727

10

Modest amyloid deposition is associated with iron dysregulation, microglial activation, and oxidative stress.

Gallagher JJ, Finnegan ME, Grehan B, Dobson J, Collingwood JF, Lynch MA. Gallagher JJ, et al. Among authors: collingwood jf. J Alzheimers Dis. 2012;28(1):147-61. doi: 10.3233/JAD-2011-110614. J Alzheimers Dis. 2012. PMID: 21971404

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