Shi G - Search Results

1

Unlike Th1, Th17 cells mediate sustained autoimmune inflammation and are highly resistant to restimulation-induced cell death.

Shi G, Ramaswamy M, Vistica BP, Cox CA, Tan C, Wawrousek EF, Siegel RM, Gery I. Shi G, et al. J Immunol. 2009 Dec 1;183(11):7547-56. doi: 10.4049/jimmunol.0900519. Epub 2009 Nov 4. J Immunol. 2009. PMID: 19890052 Free PMC article.

2

A suppressive oligodeoxynucleotide inhibits ocular inflammation.

Fujimoto C, Klinman DM, Shi G, Yin H, Vistica BP, Lovaas JD, Wawrousek EF, Igarashi T, Chan CC, Gery I. Fujimoto C, et al. Among authors: shi g. Clin Exp Immunol. 2009 Jun;156(3):528-34. doi: 10.1111/j.1365-2249.2009.03918.x. Clin Exp Immunol. 2009. PMID: 19438607 Free PMC article.

3

Phenotypes of Th lineages generated by the commonly used activation with anti-CD3/CD28 antibodies differ from those generated by the physiological activation with the specific antigen.

Tan C, Wei L, Vistica BP, Shi G, Wawrousek EF, Gery I. Tan C, et al. Among authors: shi g. Cell Mol Immunol. 2014 May;11(3):305-13. doi: 10.1038/cmi.2014.8. Epub 2014 Mar 3. Cell Mol Immunol. 2014. PMID: 24583715 Free PMC article.

4

Induced regulatory T-cells (iTregs) generated by activation with anti-CD3/CD28 antibodies differ from those generated by the physiological-like activation with antigen/APC.

Zhao C, Shi G, Vistica BP, Hinshaw SJ, Wandu WS, Tan C, Zhang M, Gery I. Zhao C, et al. Among authors: shi g. Cell Immunol. 2014 Aug;290(2):179-84. doi: 10.1016/j.cellimm.2014.06.004. Epub 2014 Jun 27. Cell Immunol. 2014. PMID: 25038545

5

Pertussis toxin is superior to TLR ligands in enhancing pathogenic autoimmunity, targeted at a neo-self antigen, by triggering robust expansion of Th1 cells and their cytokine production.

Fujimoto C, Yu CR, Shi G, Vistica BP, Wawrousek EF, Klinman DM, Chan CC, Egwuagu CE, Gery I. Fujimoto C, et al. Among authors: shi g. J Immunol. 2006 Nov 15;177(10):6896-903. doi: 10.4049/jimmunol.177.10.6896. J Immunol. 2006. PMID: 17082604

6

Microbial products trigger autoimmune ocular inflammation.

Fujimoto C, Shi G, Gery I. Fujimoto C, et al. Among authors: shi g. Ophthalmic Res. 2008;40(3-4):193-9. doi: 10.1159/000119875. Epub 2008 Apr 18. Ophthalmic Res. 2008. PMID: 18421238 Free PMC article.

7

Both Th1 and Th17 are immunopathogenic but differ in other key biological activities.

Cox CA, Shi G, Yin H, Vistica BP, Wawrousek EF, Chan CC, Gery I. Cox CA, et al. Among authors: shi g. J Immunol. 2008 Jun 1;180(11):7414-22. doi: 10.4049/jimmunol.180.11.7414. J Immunol. 2008. PMID: 18490741 Free PMC article.

8

Phenotype switching by inflammation-inducing polarized Th17 cells, but not by Th1 cells.

Shi G, Cox CA, Vistica BP, Tan C, Wawrousek EF, Gery I. Shi G, et al. J Immunol. 2008 Nov 15;181(10):7205-13. doi: 10.4049/jimmunol.181.10.7205. J Immunol. 2008. PMID: 18981142 Free PMC article.

9

Antigen-specific Th9 cells exhibit uniqueness in their kinetics of cytokine production and short retention at the inflammatory site.

Tan C, Aziz MK, Lovaas JD, Vistica BP, Shi G, Wawrousek EF, Gery I. Tan C, et al. Among authors: shi g. J Immunol. 2010 Dec 1;185(11):6795-801. doi: 10.4049/jimmunol.1001676. Epub 2010 Oct 22. J Immunol. 2010. PMID: 20971929 Free PMC article.

10

Inflammation-inducing Th1 and Th17 cells differ in their expression patterns of apoptosis-related molecules.

Tan C, Ramaswamy M, Shi G, Vistica BP, Siegel RM, Gery I. Tan C, et al. Among authors: shi g. Cell Immunol. 2011;271(2):210-3. doi: 10.1016/j.cellimm.2011.08.006. Epub 2011 Aug 17. Cell Immunol. 2011. PMID: 21903206 Free PMC article.

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