Tan C - Search Results

1

Unlike Th1, Th17 cells mediate sustained autoimmune inflammation and are highly resistant to restimulation-induced cell death.

Shi G, Ramaswamy M, Vistica BP, Cox CA, Tan C, Wawrousek EF, Siegel RM, Gery I. Shi G, et al. Among authors: tan c. J Immunol. 2009 Dec 1;183(11):7547-56. doi: 10.4049/jimmunol.0900519. Epub 2009 Nov 4. J Immunol. 2009. PMID: 19890052 Free PMC article.

2

Phenotype switching by inflammation-inducing polarized Th17 cells, but not by Th1 cells.

Shi G, Cox CA, Vistica BP, Tan C, Wawrousek EF, Gery I. Shi G, et al. Among authors: tan c. J Immunol. 2008 Nov 15;181(10):7205-13. doi: 10.4049/jimmunol.181.10.7205. J Immunol. 2008. PMID: 18981142 Free PMC article.

3

Antigen-specific Th9 cells exhibit uniqueness in their kinetics of cytokine production and short retention at the inflammatory site.

Tan C, Aziz MK, Lovaas JD, Vistica BP, Shi G, Wawrousek EF, Gery I. Tan C, et al. J Immunol. 2010 Dec 1;185(11):6795-801. doi: 10.4049/jimmunol.1001676. Epub 2010 Oct 22. J Immunol. 2010. PMID: 20971929 Free PMC article.

4

Inflammation-inducing Th1 and Th17 cells differ in their expression patterns of apoptosis-related molecules.

Tan C, Ramaswamy M, Shi G, Vistica BP, Siegel RM, Gery I. Tan C, et al. Cell Immunol. 2011;271(2):210-3. doi: 10.1016/j.cellimm.2011.08.006. Epub 2011 Aug 17. Cell Immunol. 2011. PMID: 21903206 Free PMC article.

5

The unique features of Th9 cells and their products.

Tan C, Gery I. Tan C, et al. Crit Rev Immunol. 2012;32(1):1-10. doi: 10.1615/critrevimmunol.v32.i1.10. Crit Rev Immunol. 2012. PMID: 22428852 Free PMC article. Review.

6

Cell-cell interaction with APC, not IL-23, is required for naive CD4 cells to acquire pathogenicity during Th17 lineage commitment.

Shi G, Lovaas JD, Tan C, Vistica BP, Wawrousek EF, Aziz MK, Rigden RC, Caspi RR, Gery I. Shi G, et al. Among authors: tan c. J Immunol. 2012 Aug 1;189(3):1220-7. doi: 10.4049/jimmunol.1103033. Epub 2012 Jun 27. J Immunol. 2012. PMID: 22745380 Free PMC article.

7

SLAT/Def6 plays a critical role in the pathogenic process of experimental autoimmune uveitis (EAU).

Vistica BP, Shi G, Nugent L, Tan C, Altman A, Gery I. Vistica BP, et al. Among authors: tan c. Mol Vis. 2012;18:1858-64. Epub 2012 Jul 7. Mol Vis. 2012. PMID: 22815639 Free PMC article.

8

Differential involvement of Th1 and Th17 in pathogenic autoimmune processes triggered by different TLR ligands.

Shi G, Vistica BP, Nugent LF, Tan C, Wawrousek EF, Klinman DM, Gery I. Shi G, et al. Among authors: tan c. J Immunol. 2013 Jul 1;191(1):415-23. doi: 10.4049/jimmunol.1201732. Epub 2013 May 29. J Immunol. 2013. PMID: 23720812 Free PMC article.

9

Phenotypes of Th lineages generated by the commonly used activation with anti-CD3/CD28 antibodies differ from those generated by the physiological activation with the specific antigen.

Tan C, Wei L, Vistica BP, Shi G, Wawrousek EF, Gery I. Tan C, et al. Cell Mol Immunol. 2014 May;11(3):305-13. doi: 10.1038/cmi.2014.8. Epub 2014 Mar 3. Cell Mol Immunol. 2014. PMID: 24583715 Free PMC article.

10

Induced regulatory T-cells (iTregs) generated by activation with anti-CD3/CD28 antibodies differ from those generated by the physiological-like activation with antigen/APC.

Zhao C, Shi G, Vistica BP, Hinshaw SJ, Wandu WS, Tan C, Zhang M, Gery I. Zhao C, et al. Among authors: tan c. Cell Immunol. 2014 Aug;290(2):179-84. doi: 10.1016/j.cellimm.2014.06.004. Epub 2014 Jun 27. Cell Immunol. 2014. PMID: 25038545

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