Aviram M - Search Results

1

High glucose stimulates macrophage SR-BI expression and induces a switch in its activity from cholesterol efflux to cholesterol influx.

Gantman A, Fuhrman B, Aviram M, Hayek T. Gantman A, et al. Among authors: aviram m. Biochem Biophys Res Commun. 2010 Jan 1;391(1):523-8. doi: 10.1016/j.bbrc.2009.11.091. Epub 2009 Nov 23. Biochem Biophys Res Commun. 2010. PMID: 19941833

2

Acetylcholine esterase protects LDL against oxidation.

Fuhrman B, Partoush A, Aviram M. Fuhrman B, et al. Among authors: aviram m. Biochem Biophys Res Commun. 2004 Sep 24;322(3):974-8. doi: 10.1016/j.bbrc.2004.08.019. Biochem Biophys Res Commun. 2004. PMID: 15336559

3

A biphasic U-shape effect of cellular oxidative stress on the macrophage anti-oxidant paraoxonase 2 (PON2) enzymatic activity.

Shiner M, Fuhrman B, Aviram M. Shiner M, et al. Among authors: aviram m. Biochem Biophys Res Commun. 2006 Oct 27;349(3):1094-9. doi: 10.1016/j.bbrc.2006.08.150. Epub 2006 Sep 1. Biochem Biophys Res Commun. 2006. PMID: 16970920

4

High glucose concentration increases macrophage cholesterol biosynthesis in diabetes through activation of the sterol regulatory element binding protein 1 (SREBP1): inhibitory effect of insulin.

Kaplan M, Kerry R, Aviram M, Hayek T. Kaplan M, et al. Among authors: aviram m. J Cardiovasc Pharmacol. 2008 Oct;52(4):324-32. doi: 10.1097/FJC.0b013e3181879d98. J Cardiovasc Pharmacol. 2008. PMID: 18791464

5

Paraoxonase 2 (PON2) decreases high glucose-induced macrophage triglycerides (TG) accumulation, via inhibition of NADPH-oxidase and DGAT1 activity: studies in PON2-deficient mice.

Meilin E, Aviram M, Hayek T. Meilin E, et al. Among authors: aviram m. Atherosclerosis. 2010 Feb;208(2):390-5. doi: 10.1016/j.atherosclerosis.2009.07.057. Epub 2009 Aug 8. Atherosclerosis. 2010. PMID: 19748094

6

Paraoxonase 1 (PON1) deficiency in mice is associated with reduced expression of macrophage SR-BI and consequently the loss of HDL cytoprotection against apoptosis.

Fuhrman B, Gantman A, Aviram M. Fuhrman B, et al. Among authors: aviram m. Atherosclerosis. 2010 Jul;211(1):61-8. doi: 10.1016/j.atherosclerosis.2010.01.025. Epub 2010 Jan 28. Atherosclerosis. 2010. PMID: 20149374

7

Metformin inhibits macrophage cholesterol biosynthesis rate: possible role for metformin-induced oxidative stress.

Koren-Gluzer M, Aviram M, Hayek T. Koren-Gluzer M, et al. Among authors: aviram m. Biochem Biophys Res Commun. 2013 Sep 27;439(3):396-400. doi: 10.1016/j.bbrc.2013.08.062. Epub 2013 Aug 28. Biochem Biophys Res Commun. 2013. PMID: 23994636

8

Role of HDL apolipoprotein E in cellular cholesterol efflux: studies in apo E knockout transgenic mice.

Hayek T, Oiknine J, Brook JG, Aviram M. Hayek T, et al. Among authors: aviram m. Biochem Biophys Res Commun. 1994 Dec 15;205(2):1072-8. doi: 10.1006/bbrc.1994.2775. Biochem Biophys Res Commun. 1994. PMID: 7802634

9

Hypocholesterolemic effect of lycopene and beta-carotene is related to suppression of cholesterol synthesis and augmentation of LDL receptor activity in macrophages.

Fuhrman B, Elis A, Aviram M. Fuhrman B, et al. Among authors: aviram m. Biochem Biophys Res Commun. 1997 Apr 28;233(3):658-62. doi: 10.1006/bbrc.1997.6520. Biochem Biophys Res Commun. 1997. PMID: 9168909

10

Losartan inhibits cellular uptake of oxidized LDL by monocyte-macrophages from hypercholesterolemic patients.

Hayek T, Aviram M, Heinrich R, Sakhnini E, Keidar S. Hayek T, et al. Among authors: aviram m. Biochem Biophys Res Commun. 2000 Jul 5;273(2):417-20. doi: 10.1006/bbrc.2000.2963. Biochem Biophys Res Commun. 2000. PMID: 10873620

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