González-Périz A - Search Results

1

5-lipoxygenase deficiency reduces hepatic inflammation and tumor necrosis factor alpha-induced hepatocyte damage in hyperlipidemia-prone ApoE-null mice.

Martínez-Clemente M, Ferré N, González-Périz A, López-Parra M, Horrillo R, Titos E, Morán-Salvador E, Miquel R, Arroyo V, Funk CD, Clària J. Martínez-Clemente M, et al. Hepatology. 2010 Mar;51(3):817-27. doi: 10.1002/hep.23463. Hepatology. 2010. PMID: 20112424

2

Inhibition of 5-lipoxygenase-activating protein abrogates experimental liver injury: role of Kupffer cells.

Titos E, Clària J, Planagumà A, López-Parra M, González-Périz A, Gaya J, Miquel R, Arroyo V, Rodés J. Titos E, et al. J Leukoc Biol. 2005 Oct;78(4):871-8. doi: 10.1189/jlb.1204747. Epub 2005 Jul 20. J Leukoc Biol. 2005. PMID: 16033810

3

Comparative protection against liver inflammation and fibrosis by a selective cyclooxygenase-2 inhibitor and a nonredox-type 5-lipoxygenase inhibitor.

Horrillo R, Planagumà A, González-Périz A, Ferré N, Titos E, Miquel R, López-Parra M, Masferrer JL, Arroyo V, Clària J. Horrillo R, et al. J Pharmacol Exp Ther. 2007 Dec;323(3):778-86. doi: 10.1124/jpet.107.128264. Epub 2007 Aug 31. J Pharmacol Exp Ther. 2007. PMID: 17766677

4

Regulatory effects of arachidonate 5-lipoxygenase on hepatic microsomal TG transfer protein activity and VLDL-triglyceride and apoB secretion in obese mice.

López-Parra M, Titos E, Horrillo R, Ferré N, González-Périz A, Martínez-Clemente M, Planagumà A, Masferrer J, Arroyo V, Clària J. López-Parra M, et al. J Lipid Res. 2008 Dec;49(12):2513-23. doi: 10.1194/jlr.M800101-JLR200. Epub 2008 Jul 21. J Lipid Res. 2008. PMID: 18645210 Free article.

5

5-lipoxygenase activating protein signals adipose tissue inflammation and lipid dysfunction in experimental obesity.

Horrillo R, González-Périz A, Martínez-Clemente M, López-Parra M, Ferré N, Titos E, Morán-Salvador E, Deulofeu R, Arroyo V, Clària J. Horrillo R, et al. J Immunol. 2010 Apr 1;184(7):3978-87. doi: 10.4049/jimmunol.0901355. Epub 2010 Mar 5. J Immunol. 2010. PMID: 20207999

6

Disruption of the 12/15-lipoxygenase gene (Alox15) protects hyperlipidemic mice from nonalcoholic fatty liver disease.

Martínez-Clemente M, Ferré N, Titos E, Horrillo R, González-Périz A, Morán-Salvador E, López-Vicario C, Miquel R, Arroyo V, Funk CD, Clària J. Martínez-Clemente M, et al. Hepatology. 2010 Dec;52(6):1980-91. doi: 10.1002/hep.23928. Epub 2010 Oct 21. Hepatology. 2010. PMID: 20967760

7

Role for PPARγ in obesity-induced hepatic steatosis as determined by hepatocyte- and macrophage-specific conditional knockouts.

Morán-Salvador E, López-Parra M, García-Alonso V, Titos E, Martínez-Clemente M, González-Périz A, López-Vicario C, Barak Y, Arroyo V, Clària J. Morán-Salvador E, et al. Among authors: gonzalez periz a. FASEB J. 2011 Aug;25(8):2538-50. doi: 10.1096/fj.10-173716. Epub 2011 Apr 19. FASEB J. 2011. PMID: 21507897

8

Resolvin D1 and its precursor docosahexaenoic acid promote resolution of adipose tissue inflammation by eliciting macrophage polarization toward an M2-like phenotype.

Titos E, Rius B, González-Périz A, López-Vicario C, Morán-Salvador E, Martínez-Clemente M, Arroyo V, Clària J. Titos E, et al. Among authors: gonzalez periz a. J Immunol. 2011 Nov 15;187(10):5408-18. doi: 10.4049/jimmunol.1100225. Epub 2011 Oct 17. J Immunol. 2011. PMID: 22013115

9

Cell-specific PPARγ deficiency establishes anti-inflammatory and anti-fibrogenic properties for this nuclear receptor in non-parenchymal liver cells.

Morán-Salvador E, Titos E, Rius B, González-Périz A, García-Alonso V, López-Vicario C, Miquel R, Barak Y, Arroyo V, Clària J. Morán-Salvador E, et al. Among authors: gonzalez periz a. J Hepatol. 2013 Nov;59(5):1045-53. doi: 10.1016/j.jhep.2013.06.023. Epub 2013 Jul 2. J Hepatol. 2013. PMID: 23831119

10

New approaches to the modulation of the cyclooxygenase-2 and 5-lipoxygenase pathways.

González-Périz A, Clària J. González-Périz A, et al. Curr Top Med Chem. 2007;7(3):297-309. doi: 10.2174/156802607779941378. Curr Top Med Chem. 2007. PMID: 17305572 Review.

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