de Wit C - Search Results

1

Amplification of EDHF-type vasodilatations in TRPC1-deficient mice.

Schmidt K, Dubrovska G, Nielsen G, Fesüs G, Uhrenholt TR, Hansen PB, Gudermann T, Dietrich A, Gollasch M, de Wit C, Köhler R. Schmidt K, et al. Among authors: de wit c. Br J Pharmacol. 2010 Dec;161(8):1722-33. doi: 10.1111/j.1476-5381.2010.00985.x. Br J Pharmacol. 2010. PMID: 20718731 Free PMC article.

2

Activation of KCa3.1 by SKA-31 induces arteriolar dilatation and lowers blood pressure in normo- and hypertensive connexin40-deficient mice.

Radtke J, Schmidt K, Wulff H, Köhler R, de Wit C. Radtke J, et al. Among authors: de wit c. Br J Pharmacol. 2013 Sep;170(2):293-303. doi: 10.1111/bph.12267. Br J Pharmacol. 2013. PMID: 23734697 Free PMC article.

3

Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo.

Wölfle SE, Schmidt VJ, Hoyer J, Köhler R, de Wit C. Wölfle SE, et al. Among authors: de wit c. Cardiovasc Res. 2009 Jun 1;82(3):476-83. doi: 10.1093/cvr/cvp060. Epub 2009 Feb 13. Cardiovasc Res. 2009. PMID: 19218287

4

Crucial importance of the endothelial K+ channel SK3 and connexin40 in arteriolar dilations during skeletal muscle contraction.

Milkau M, Köhler R, de Wit C. Milkau M, et al. Among authors: de wit c. FASEB J. 2010 Sep;24(9):3572-9. doi: 10.1096/fj.10-158956. Epub 2010 Apr 28. FASEB J. 2010. PMID: 20427707

5

Pharmacological activation of KCa3.1/KCa2.3 channels produces endothelial hyperpolarization and lowers blood pressure in conscious dogs.

Damkjaer M, Nielsen G, Bodendiek S, Staehr M, Gramsbergen JB, de Wit C, Jensen BL, Simonsen U, Bie P, Wulff H, Köhler R. Damkjaer M, et al. Among authors: de wit c. Br J Pharmacol. 2012 Jan;165(1):223-34. doi: 10.1111/j.1476-5381.2011.01546.x. Br J Pharmacol. 2012. PMID: 21699504 Free PMC article.

6

Keep calm and carry on: miR-1298 prevents up-regulation of Cx43 and secures a quiescent vascular smooth muscle cell.

Schmidt K, de Wit C. Schmidt K, et al. Among authors: de wit c. Cardiovasc Res. 2015 Sep 1;107(4):407-9. doi: 10.1093/cvr/cvv200. Epub 2015 Jul 21. Cardiovasc Res. 2015. PMID: 26199195 No abstract available.

7

Endothelium-Derived Hyperpolarizing Factor and Myoendothelial Coupling: The in vivo Perspective.

Schmidt K, de Wit C. Schmidt K, et al. Among authors: de wit c. Front Physiol. 2020 Dec 23;11:602930. doi: 10.3389/fphys.2020.602930. eCollection 2020. Front Physiol. 2020. PMID: 33424626 Free PMC article. Review.

8

Communication Through Gap Junctions in the Endothelium.

Schmidt K, Windler R, de Wit C. Schmidt K, et al. Among authors: de wit c. Adv Pharmacol. 2016;77:209-40. doi: 10.1016/bs.apha.2016.04.004. Epub 2016 May 9. Adv Pharmacol. 2016. PMID: 27451099

9

Defective Cx40 maintains Cx37 expression but intact Cx40 is crucial for conducted dilations irrespective of hypertension.

Jobs A, Schmidt K, Schmidt VJ, Lübkemeier I, van Veen TA, Kurtz A, Willecke K, de Wit C. Jobs A, et al. Among authors: de wit c. Hypertension. 2012 Dec;60(6):1422-9. doi: 10.1161/HYPERTENSIONAHA.112.201194. Epub 2012 Oct 22. Hypertension. 2012. PMID: 23090768

10

Impaired endothelium-derived hyperpolarizing factor-mediated dilations and increased blood pressure in mice deficient of the intermediate-conductance Ca2+-activated K+ channel.

Si H, Heyken WT, Wölfle SE, Tysiac M, Schubert R, Grgic I, Vilianovich L, Giebing G, Maier T, Gross V, Bader M, de Wit C, Hoyer J, Köhler R. Si H, et al. Among authors: de wit c. Circ Res. 2006 Sep 1;99(5):537-44. doi: 10.1161/01.RES.0000238377.08219.0c. Epub 2006 Jul 27. Circ Res. 2006. PMID: 16873714

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