Worman HJ - Search Results

1

Lamin A variants that cause striated muscle disease are defective in anchoring transmembrane actin-associated nuclear lines for nuclear movement.

Folker ES, Ostlund C, Luxton GW, Worman HJ, Gundersen GG. Folker ES, et al. Among authors: worman hj. Proc Natl Acad Sci U S A. 2011 Jan 4;108(1):131-6. doi: 10.1073/pnas.1000824108. Epub 2010 Dec 20. Proc Natl Acad Sci U S A. 2011. PMID: 21173262 Free PMC article.

2

A lamin B receptor in the nuclear envelope.

Worman HJ, Yuan J, Blobel G, Georgatos SD. Worman HJ, et al. Proc Natl Acad Sci U S A. 1988 Nov;85(22):8531-4. doi: 10.1073/pnas.85.22.8531. Proc Natl Acad Sci U S A. 1988. PMID: 2847165 Free PMC article.

3

Intracellular trafficking of emerin, the Emery-Dreifuss muscular dystrophy protein.

Ostlund C, Ellenberg J, Hallberg E, Lippincott-Schwartz J, Worman HJ. Ostlund C, et al. Among authors: worman hj. J Cell Sci. 1999 Jun;112 ( Pt 11):1709-19. doi: 10.1242/jcs.112.11.1709. J Cell Sci. 1999. PMID: 10318763

4

Transcriptional repression of euchromatic genes by Drosophila heterochromatin protein 1 and histone modifiers.

Hwang KK, Eissenberg JC, Worman HJ. Hwang KK, et al. Among authors: worman hj. Proc Natl Acad Sci U S A. 2001 Sep 25;98(20):11423-7. doi: 10.1073/pnas.211303598. Epub 2001 Sep 18. Proc Natl Acad Sci U S A. 2001. PMID: 11562500 Free PMC article.

5

Properties of lamin A mutants found in Emery-Dreifuss muscular dystrophy, cardiomyopathy and Dunnigan-type partial lipodystrophy.

Ostlund C, Bonne G, Schwartz K, Worman HJ. Ostlund C, et al. Among authors: worman hj. J Cell Sci. 2001 Dec;114(Pt 24):4435-45. doi: 10.1242/jcs.114.24.4435. J Cell Sci. 2001. PMID: 11792809

6

1H, 13C and 15N resonance assignments of the C-terminal domain of human lamin A/C.

Krimm I, Couprie J, Ostlund C, Worman HJ, Zinn-Justin S. Krimm I, et al. Among authors: worman hj. J Biomol NMR. 2002 Apr;22(4):371-2. doi: 10.1023/a:1014943122068. J Biomol NMR. 2002. PMID: 12018485 No abstract available.

7

The Ig-like structure of the C-terminal domain of lamin A/C, mutated in muscular dystrophies, cardiomyopathy, and partial lipodystrophy.

Krimm I, Ostlund C, Gilquin B, Couprie J, Hossenlopp P, Mornon JP, Bonne G, Courvalin JC, Worman HJ, Zinn-Justin S. Krimm I, et al. Among authors: worman hj. Structure. 2002 Jun;10(6):811-23. doi: 10.1016/s0969-2126(02)00777-3. Structure. 2002. PMID: 12057196 Free article.

8

Expression of lamin A mutated in the carboxyl-terminal tail generates an aberrant nuclear phenotype similar to that observed in cells from patients with Dunnigan-type partial lipodystrophy and Emery-Dreifuss muscular dystrophy.

Favreau C, Dubosclard E, Ostlund C, Vigouroux C, Capeau J, Wehnert M, Higuet D, Worman HJ, Courvalin JC, Buendia B. Favreau C, et al. Among authors: worman hj. Exp Cell Res. 2003 Jan 1;282(1):14-23. doi: 10.1006/excr.2002.5669. Exp Cell Res. 2003. PMID: 12490190

9

The nuclear lamina and inherited disease.

Worman HJ, Courvalin JC. Worman HJ, et al. Trends Cell Biol. 2002 Dec;12(12):591-8. doi: 10.1016/s0962-8924(02)02401-7. Trends Cell Biol. 2002. PMID: 12495848 Review.

10

Nuclear envelope proteins and neuromuscular diseases.

Ostlund C, Worman HJ. Ostlund C, et al. Among authors: worman hj. Muscle Nerve. 2003 Apr;27(4):393-406. doi: 10.1002/mus.10302. Muscle Nerve. 2003. PMID: 12661041 Review.

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