Reist NE - Search Results

1

Membrane penetration by synaptotagmin is required for coupling calcium binding to vesicle fusion in vivo.

Paddock BE, Wang Z, Biela LM, Chen K, Getzy MD, Striegel A, Richmond JE, Chapman ER, Featherstone DE, Reist NE. Paddock BE, et al. Among authors: reist ne. J Neurosci. 2011 Feb 9;31(6):2248-57. doi: 10.1523/JNEUROSCI.3153-09.2011. J Neurosci. 2011. PMID: 21307261 Free PMC article.

2

Ca2+-dependent, phospholipid-binding residues of synaptotagmin are critical for excitation-secretion coupling in vivo.

Paddock BE, Striegel AR, Hui E, Chapman ER, Reist NE. Paddock BE, et al. Among authors: reist ne. J Neurosci. 2008 Jul 23;28(30):7458-66. doi: 10.1523/JNEUROSCI.0197-08.2008. J Neurosci. 2008. PMID: 18650324 Free PMC article.

3

Synaptotagmin I stabilizes synaptic vesicles via its C(2)A polylysine motif.

Mace KE, Biela LM, Sares AG, Reist NE. Mace KE, et al. Among authors: reist ne. Genesis. 2009 May;47(5):337-45. doi: 10.1002/dvg.20502. Genesis. 2009. PMID: 19358157

4

Calcium binding by synaptotagmin's C2A domain is an essential element of the electrostatic switch that triggers synchronous synaptic transmission.

Striegel AR, Biela LM, Evans CS, Wang Z, Delehoy JB, Sutton RB, Chapman ER, Reist NE. Striegel AR, et al. Among authors: reist ne. J Neurosci. 2012 Jan 25;32(4):1253-60. doi: 10.1523/JNEUROSCI.4652-11.2012. J Neurosci. 2012. PMID: 22279210 Free PMC article.

5

Morphologically docked synaptic vesicles are reduced in synaptotagmin mutants of Drosophila.

Reist NE, Buchanan J, Li J, DiAntonio A, Buxton EM, Schwarz TL. Reist NE, et al. J Neurosci. 1998 Oct 1;18(19):7662-73. doi: 10.1523/JNEUROSCI.18-19-07662.1998. J Neurosci. 1998. PMID: 9742137 Free PMC article.

6

Synaptotagmin: Mechanisms of an electrostatic switch.

Bowers MR, Reist NE. Bowers MR, et al. Among authors: reist ne. Neurosci Lett. 2020 Mar 23;722:134834. doi: 10.1016/j.neulet.2020.134834. Epub 2020 Feb 10. Neurosci Lett. 2020. PMID: 32057923 Review.

7

The C(2)B Ca(2+)-binding motif of synaptotagmin is required for synaptic transmission in vivo.

Mackler JM, Drummond JA, Loewen CA, Robinson IM, Reist NE. Mackler JM, et al. Among authors: reist ne. Nature. 2002 Jul 18;418(6895):340-4. doi: 10.1038/nature00846. Epub 2002 Jul 7. Nature. 2002. PMID: 12110842

8

Drosophila synaptotagmin I null mutants show severe alterations in vesicle populations but calcium-binding motif mutants do not.

Loewen CA, Royer SM, Reist NE. Loewen CA, et al. Among authors: reist ne. J Comp Neurol. 2006 May 1;496(1):1-12. doi: 10.1002/cne.20868. J Comp Neurol. 2006. PMID: 16528727

9

C2B polylysine motif of synaptotagmin facilitates a Ca2+-independent stage of synaptic vesicle priming in vivo.

Loewen CA, Lee SM, Shin YK, Reist NE. Loewen CA, et al. Among authors: reist ne. Mol Biol Cell. 2006 Dec;17(12):5211-26. doi: 10.1091/mbc.e06-07-0622. Epub 2006 Sep 20. Mol Biol Cell. 2006. PMID: 16987956 Free PMC article.

10

The C2A domain of synaptotagmin is an essential component of the calcium sensor for synaptic transmission.

Bowers MR, Reist NE. Bowers MR, et al. Among authors: reist ne. PLoS One. 2020 Feb 7;15(2):e0228348. doi: 10.1371/journal.pone.0228348. eCollection 2020. PLoS One. 2020. PMID: 32032373 Free PMC article.

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