Olesen SP - Search Results

1

Physiological consequences of transient outward K+ current activation during heart failure in the canine left ventricle.

Cordeiro JM, Calloe K, Moise NS, Kornreich B, Giannandrea D, Di Diego JM, Olesen SP, Antzelevitch C. Cordeiro JM, et al. Among authors: olesen sp. J Mol Cell Cardiol. 2012 Jun;52(6):1291-8. doi: 10.1016/j.yjmcc.2012.03.001. Epub 2012 Mar 11. J Mol Cell Cardiol. 2012. PMID: 22434032 Free PMC article.

2

A transient outward potassium current activator recapitulates the electrocardiographic manifestations of Brugada syndrome.

Calloe K, Cordeiro JM, Di Diego JM, Hansen RS, Grunnet M, Olesen SP, Antzelevitch C. Calloe K, et al. Among authors: olesen sp. Cardiovasc Res. 2009 Mar 1;81(4):686-94. doi: 10.1093/cvr/cvn339. Epub 2008 Dec 10. Cardiovasc Res. 2009. PMID: 19073629 Free PMC article.

3

Differential effects of the transient outward K(+) current activator NS5806 in the canine left ventricle.

Calloe K, Soltysinska E, Jespersen T, Lundby A, Antzelevitch C, Olesen SP, Cordeiro JM. Calloe K, et al. Among authors: olesen sp. J Mol Cell Cardiol. 2010 Jan;48(1):191-200. doi: 10.1016/j.yjmcc.2009.07.017. Epub 2009 Jul 24. J Mol Cell Cardiol. 2010. PMID: 19632239 Free PMC article.

4

Comparison of the effects of a transient outward potassium channel activator on currents recorded from atrial and ventricular cardiomyocytes.

Calloe K, Nof E, Jespersen T, Di Diego JM, Chlus N, Olesen SP, Antzelevitch C, Cordeiro JM. Calloe K, et al. Among authors: olesen sp. J Cardiovasc Electrophysiol. 2011 Sep;22(9):1057-66. doi: 10.1111/j.1540-8167.2011.02053.x. Epub 2011 Apr 1. J Cardiovasc Electrophysiol. 2011. PMID: 21457383 Free PMC article.

5

Tissue-specific effects of acetylcholine in the canine heart.

Calloe K, Goodrow R, Olesen SP, Antzelevitch C, Cordeiro JM. Calloe K, et al. Among authors: olesen sp. Am J Physiol Heart Circ Physiol. 2013 Jul 1;305(1):H66-75. doi: 10.1152/ajpheart.00029.2013. Epub 2013 May 3. Am J Physiol Heart Circ Physiol. 2013. PMID: 23645460 Free PMC article.

6

Development of heart failure is independent of K+ channel-interacting protein 2 expression.

Speerschneider T, Grubb S, Metoska A, Olesen SP, Calloe K, Thomsen MB. Speerschneider T, et al. Among authors: olesen sp. J Physiol. 2013 Dec 1;591(23):5923-37. doi: 10.1113/jphysiol.2013.263483. Epub 2013 Oct 7. J Physiol. 2013. PMID: 24099801 Free PMC article.

7

Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice.

Grubb S, Speerschneider T, Occhipinti D, Fiset C, Olesen SP, Thomsen MB, Calloe K. Grubb S, et al. Among authors: olesen sp. J Cardiovasc Electrophysiol. 2014 Aug;25(8):896-904. doi: 10.1111/jce.12422. Epub 2014 May 2. J Cardiovasc Electrophysiol. 2014. PMID: 24678923

8

The small molecule NS11021 is a potent and specific activator of Ca2+-activated big-conductance K+ channels.

Bentzen BH, Nardi A, Calloe K, Madsen LS, Olesen SP, Grunnet M. Bentzen BH, et al. Among authors: olesen sp. Mol Pharmacol. 2007 Oct;72(4):1033-44. doi: 10.1124/mol.107.038331. Epub 2007 Jul 16. Mol Pharmacol. 2007. PMID: 17636045

9

Ventricular repolarization time, location of pacing stimulus and current pulse amplitude conspire to determine arrhythmogenicity in mice.

Speerschneider T, Grubb S, Olesen SP, Calloe K, Thomsen MB. Speerschneider T, et al. Among authors: olesen sp. Acta Physiol (Oxf). 2017 Mar;219(3):660-668. doi: 10.1111/apha.12761. Epub 2016 Aug 24. Acta Physiol (Oxf). 2017. PMID: 27459728

10

Functional assessment of compound mutations in the KCNQ1 and KCNH2 genes associated with long QT syndrome.

Grunnet M, Behr ER, Calloe K, Hofman-Bang J, Till J, Christiansen M, McKenna WJ, Olesen SP, Schmitt N. Grunnet M, et al. Among authors: olesen sp. Heart Rhythm. 2005 Nov;2(11):1238-49. doi: 10.1016/j.hrthm.2005.07.025. Heart Rhythm. 2005. PMID: 16253915

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