Oancea C - Search Results

1

Deacetylase inhibitors modulate proliferation and self-renewal properties of leukemic stem and progenitor cells.

Romanski A, Schwarz K, Keller M, Wietbrauk S, Vogel A, Roos J, Oancea C, Brill B, Krämer OH, Serve H, Ruthardt M, Bug G. Romanski A, et al. Among authors: oancea c. Cell Cycle. 2012 Sep 1;11(17):3219-26. doi: 10.4161/cc.21565. Epub 2012 Aug 16. Cell Cycle. 2012. PMID: 22895185 Free PMC article.

2

Cooperation between constitutively activated c-Kit signaling and leukemogenic transcription factors in the determination of the leukemic phenotype in murine hematopoietic stem cells.

Zheng X, Oancea C, Henschler R, Ruthardt M. Zheng X, et al. Among authors: oancea c. Int J Oncol. 2009 Jun;34(6):1521-31. doi: 10.3892/ijo_00000281. Int J Oncol. 2009. PMID: 19424569

3

Sulindac sulfide reverses aberrant self-renewal of progenitor cells induced by the AML-associated fusion proteins PML/RARα and PLZF/RARα.

Steinert G, Oancea C, Roos J, Hagemeyer H, Maier T, Ruthardt M, Puccetti E. Steinert G, et al. Among authors: oancea c. PLoS One. 2011;6(7):e22540. doi: 10.1371/journal.pone.0022540. Epub 2011 Jul 19. PLoS One. 2011. PMID: 21811629 Free PMC article.

4

5-Lipoxygenase is a candidate target for therapeutic management of stem cell-like cells in acute myeloid leukemia.

Roos J, Oancea C, Heinssmann M, Khan D, Held H, Kahnt AS, Capelo R, la Buscató E, Proschak E, Puccetti E, Steinhilber D, Fleming I, Maier TJ, Ruthardt M. Roos J, et al. Among authors: oancea c. Cancer Res. 2014 Sep 15;74(18):5244-55. doi: 10.1158/0008-5472.CAN-13-3012. Epub 2014 Jul 31. Cancer Res. 2014. PMID: 25082812

5

STAT activation status differentiates leukemogenic from non-leukemogenic stem cells in AML and is suppressed by arsenic in t(6;9)-positive AML.

Oancea C, Rüster B, Brill B, Roos J, Heinssmann M, Bug G, Mian AA, Guillen NA, Kornblau SM, Henschler R, Ruthardt M. Oancea C, et al. Genes Cancer. 2014 Nov;5(11-12):378-92. doi: 10.18632/genesandcancer.39. Genes Cancer. 2014. PMID: 25568664 Free PMC article.

6

The t(6;9) associated DEK/CAN fusion protein targets a population of long-term repopulating hematopoietic stem cells for leukemogenic transformation.

Oancea C, Rüster B, Henschler R, Puccetti E, Ruthardt M. Oancea C, et al. Leukemia. 2010 Nov;24(11):1910-9. doi: 10.1038/leu.2010.180. Epub 2010 Sep 9. Leukemia. 2010. PMID: 20827285

7

Reciprocal t(9;22) ABL/BCR fusion proteins: leukemogenic potential and effects on B cell commitment.

Zheng X, Oancea C, Henschler R, Moore MA, Ruthardt M. Zheng X, et al. Among authors: oancea c. PLoS One. 2009 Oct 30;4(10):e7661. doi: 10.1371/journal.pone.0007661. PLoS One. 2009. PMID: 19876398 Free PMC article.

8

The functional interplay between the t(9;22)-associated fusion proteins BCR/ABL and ABL/BCR in Philadelphia chromosome-positive acute lymphatic leukemia.

Rafiei A, Mian AA, Döring C, Metodieva A, Oancea C, Thalheimer FB, Hansmann ML, Ottmann OG, Ruthardt M. Rafiei A, et al. Among authors: oancea c. PLoS Genet. 2015 Apr 28;11(4):e1005144. doi: 10.1371/journal.pgen.1005144. eCollection 2015 Apr. PLoS Genet. 2015. PMID: 25919613 Free PMC article.

9

BCR: a new target in resistance mediated by BCR/ABL-315I?

Haberbosch I, Rafiei A, Oancea C, Ottmann GO, Ruthardt M, Mian AA. Haberbosch I, et al. Among authors: oancea c. Genes Cancer. 2016 Jan;7(1-2):36-46. doi: 10.18632/genesandcancer.93. Genes Cancer. 2016. PMID: 27014420 Free PMC article.

10

The gatekeeper mutation T315I confers resistance against small molecules by increasing or restoring the ABL-kinase activity accompanied by aberrant transphosphorylation of endogenous BCR, even in loss-of-function mutants of BCR/ABL.

Mian AA, Schüll M, Zhao Z, Oancea C, Hundertmark A, Beissert T, Ottmann OG, Ruthardt M. Mian AA, et al. Among authors: oancea c. Leukemia. 2009 Sep;23(9):1614-21. doi: 10.1038/leu.2009.69. Epub 2009 Apr 16. Leukemia. 2009. PMID: 19369965

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