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DNA damage responses and oxidative stress in dyskeratosis congenita.
Pereboeva L, Westin E, Patel T, Flaniken I, Lamb L, Klingelhutz A, Goldman F. Pereboeva L, et al. Among authors: goldman f. PLoS One. 2013 Oct 4;8(10):e76473. doi: 10.1371/journal.pone.0076473. eCollection 2013. PLoS One. 2013. PMID: 24124565 Free PMC article.
Proliferative defects in dyskeratosis congenita skin keratinocytes are corrected by expression of the telomerase reverse transcriptase, TERT, or by activation of endogenous telomerase through expression of papillomavirus E6/E7 or the telomerase RNA component, TERC.
Gourronc FA, Robertson mM, Herrig AK, Lansdorp PM, Goldman FD, Klingelhutz AJ. Gourronc FA, et al. Exp Dermatol. 2010 Mar;19(3):279-88. doi: 10.1111/j.1600-0625.2009.00916.x. Epub 2009 Jun 23. Exp Dermatol. 2010. PMID: 19558498 Free PMC article.
158 results