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The Pseudomonas aeruginosa exoenzyme Y impairs endothelial cell proliferation and vascular repair following lung injury.
Stevens TC, Ochoa CD, Morrow KA, Robson MJ, Prasain N, Zhou C, Alvarez DF, Frank DW, Balczon R, Stevens T. Stevens TC, et al. Among authors: zhou c. Am J Physiol Lung Cell Mol Physiol. 2014 May 15;306(10):L915-24. doi: 10.1152/ajplung.00135.2013. Epub 2014 Apr 4. Am J Physiol Lung Cell Mol Physiol. 2014. PMID: 24705722 Free PMC article.
In the absence of effector proteins, the Pseudomonas aeruginosa type three secretion system needle tip complex contributes to lung injury and systemic inflammatory responses.
Audia JP, Lindsey AS, Housley NA, Ochoa CR, Zhou C, Toba M, Oka M, Annamdevula NS, Fitzgerald MS, Frank DW, Alvarez DF. Audia JP, et al. Among authors: zhou c. PLoS One. 2013 Nov 27;8(11):e81792. doi: 10.1371/journal.pone.0081792. eCollection 2013. PLoS One. 2013. PMID: 24312357 Free PMC article.
Pseudomonas aeruginosa infection liberates transmissible, cytotoxic prion amyloids.
Balczon R, Morrow KA, Zhou C, Edmonds B, Alexeyev M, Pittet JF, Wagener BM, Moser SA, Leavesley S, Zha X, Frank DW, Stevens T. Balczon R, et al. Among authors: zhou c. FASEB J. 2017 Jul;31(7):2785-2796. doi: 10.1096/fj.201601042RR. Epub 2017 Mar 17. FASEB J. 2017. PMID: 28314768 Free PMC article.
Demonstration of long-lived cytotoxic agents after Pseudomonas infection may establish a molecular link to the high rates of death as a result of end-organ damage in the months after recovery from pneumonia, and modulation of signal transduction pathways that have been linked to …
Demonstration of long-lived cytotoxic agents after Pseudomonas infection may establish a molecular link to the high rates of death as a resu …
21,197 results
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