Huganir RL - Search Results

1

Postsynaptic potentiation of corticospinal projecting neurons in the anterior cingulate cortex after nerve injury.

Chen T, Koga K, Descalzi G, Qiu S, Wang J, Zhang LS, Zhang ZJ, He XB, Qin X, Xu FQ, Hu J, Wei F, Huganir RL, Li YQ, Zhuo M. Chen T, et al. Among authors: huganir rl. Mol Pain. 2014 Jun 3;10:33. doi: 10.1186/1744-8069-10-33. Mol Pain. 2014. PMID: 24890933 Free PMC article.

2

Impaired NMDA receptor-mediated postsynaptic function and blunted NMDA receptor-dependent persistent pain in mice lacking postsynaptic density-93 protein.

Tao YX, Rumbaugh G, Wang GD, Petralia RS, Zhao C, Kauer FW, Tao F, Zhuo M, Wenthold RJ, Raja SN, Huganir RL, Bredt DS, Johns RA. Tao YX, et al. Among authors: huganir rl. J Neurosci. 2003 Jul 30;23(17):6703-12. doi: 10.1523/JNEUROSCI.23-17-06703.2003. J Neurosci. 2003. PMID: 12890763 Free PMC article.

3

Preserved acute pain and impaired neuropathic pain in mice lacking protein interacting with C Kinase 1.

Wang W, Petralia RS, Takamiya K, Xia J, Li YQ, Huganir RL, Tao YX, Yaster M. Wang W, et al. Among authors: huganir rl. Mol Pain. 2011 Feb 3;7:11. doi: 10.1186/1744-8069-7-11. Mol Pain. 2011. PMID: 21291534 Free PMC article.

4

GluA1 phosphorylation contributes to postsynaptic amplification of neuropathic pain in the insular cortex.

Qiu S, Zhang M, Liu Y, Guo Y, Zhao H, Song Q, Zhao M, Huganir RL, Luo J, Xu H, Zhuo M. Qiu S, et al. Among authors: huganir rl. J Neurosci. 2014 Oct 1;34(40):13505-15. doi: 10.1523/JNEUROSCI.1431-14.2014. J Neurosci. 2014. PMID: 25274827 Free PMC article.

5

Postsynaptic insertion of AMPA receptor onto cortical pyramidal neurons in the anterior cingulate cortex after peripheral nerve injury.

Chen T, Wang W, Dong YL, Zhang MM, Wang J, Koga K, Liao YH, Li JL, Budisantoso T, Shigemoto R, Itakura M, Huganir RL, Li YQ, Zhuo M. Chen T, et al. Among authors: huganir rl. Mol Brain. 2014 Oct 31;7:76. doi: 10.1186/s13041-014-0076-8. Mol Brain. 2014. PMID: 25359681 Free PMC article.

6

Coexistence of two forms of LTP in ACC provides a synaptic mechanism for the interactions between anxiety and chronic pain.

Koga K, Descalzi G, Chen T, Ko HG, Lu J, Li S, Son J, Kim T, Kwak C, Huganir RL, Zhao MG, Kaang BK, Collingridge GL, Zhuo M. Koga K, et al. Among authors: huganir rl. Neuron. 2015 Jan 21;85(2):377-89. doi: 10.1016/j.neuron.2014.12.021. Epub 2014 Dec 31. Neuron. 2015. PMID: 25556835 Free PMC article.

7

Selective Phosphorylation of AMPA Receptor Contributes to the Network of Long-Term Potentiation in the Anterior Cingulate Cortex.

Song Q, Zheng HW, Li XH, Huganir RL, Kuner T, Zhuo M, Chen T. Song Q, et al. Among authors: huganir rl. J Neurosci. 2017 Aug 30;37(35):8534-8548. doi: 10.1523/JNEUROSCI.0925-17.2017. Epub 2017 Aug 1. J Neurosci. 2017. PMID: 28765333 Free PMC article.

8

Coexistence of Two Forms of LTP in ACC Provides a Synaptic Mechanism for the Interactions between Anxiety and Chronic Pain.

Koga K, Descalzi G, Chen T, Ko HG, Lu J, Li S, Son J, Kim T, Kwak C, Huganir RL, Zhao MG, Kaang BK, Collingridge GL, Zhuo M. Koga K, et al. Among authors: huganir rl. Neuron. 2015 May 20;86(4):1109. doi: 10.1016/j.neuron.2015.05.016. Epub 2015 May 20. Neuron. 2015. PMID: 28898636 Free article. No abstract available.

9

Spinal cord protein interacting with C kinase 1 is required for the maintenance of complete Freund's adjuvant-induced inflammatory pain but not for incision-induced post-operative pain.

Atianjoh FE, Yaster M, Zhao X, Takamiya K, Xia J, Gauda EB, Huganir RL, Tao YX. Atianjoh FE, et al. Among authors: huganir rl. Pain. 2010 Oct;151(1):226-234. doi: 10.1016/j.pain.2010.07.017. Epub 2010 Aug 8. Pain. 2010. PMID: 20696523 Free PMC article.

10

Identification and characterization of a novel phosphorylation site on the GluR1 subunit of AMPA receptors.

Lee HK, Takamiya K, Kameyama K, He K, Yu S, Rossetti L, Wilen D, Huganir RL. Lee HK, et al. Among authors: huganir rl. Mol Cell Neurosci. 2007 Sep;36(1):86-94. doi: 10.1016/j.mcn.2007.06.003. Epub 2007 Jun 27. Mol Cell Neurosci. 2007. PMID: 17689977 Free PMC article.

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