Conn PM - Search Results

1

Protein kinase C-mediated gonadotropin-releasing hormone receptor sequestration is associated with uncoupling of phosphoinositide hydrolysis.

Huckle WR, Hawes BE, Conn PM. Huckle WR, et al. Among authors: conn pm. J Biol Chem. 1989 May 25;264(15):8619-26. J Biol Chem. 1989. PMID: 2542281 Free article.

2

Sodium fluoride provokes gonadotrope desensitization to gonadotropin-releasing hormone (GnRH) and gonadotrope sensitization to A23187: evidence for multiple G proteins in GnRH action.

Hawes BE, Marzen JE, Waters SB, Conn PM. Hawes BE, et al. Among authors: conn pm. Endocrinology. 1992 May;130(5):2465-75. doi: 10.1210/endo.130.5.1315244. Endocrinology. 1992. PMID: 1315244

3

Selection of models for the study of GnRH stimulated gonadotropin release prejudices the assignment of roles for mediators and modulators of hormone action.

Conn PM, Hawes BE, Janovick JA. Conn PM, et al. Mol Cell Endocrinol. 1992 Apr;84(3):C33-7. doi: 10.1016/0303-7207(92)90025-2. Mol Cell Endocrinol. 1992. PMID: 1316852 Review. No abstract available.

4

Development of gonadotrope desensitization to gonadotropin-releasing hormone (GnRH) and recovery are not coupled to inositol phosphate production or GnRH receptor number.

Hawes BE, Conn PM. Hawes BE, et al. Among authors: conn pm. Endocrinology. 1992 Dec;131(6):2681-9. doi: 10.1210/endo.131.6.1332845. Endocrinology. 1992. PMID: 1332845

5

Gonadotropin-releasing hormone-stimulated intracellular Ca2+ fluctuations and luteinizing hormone release can be uncoupled from inositol phosphate production.

Hawes BE, Waters SB, Janovick JA, Bleasdale JE, Conn PM. Hawes BE, et al. Among authors: conn pm. Endocrinology. 1992 Jun;130(6):3475-83. doi: 10.1210/endo.130.6.1597151. Endocrinology. 1992. PMID: 1597151

6

Stimulation of luteinizing hormone release by sodium fluoride is independent of protein kinase-C activity and unaffected by desensitization to gonadotropin-releasing hormone.

Waters SB, Hawes BE, Conn PM. Waters SB, et al. Among authors: conn pm. Endocrinology. 1990 May;126(5):2583-91. doi: 10.1210/endo-126-5-2583. Endocrinology. 1990. PMID: 2158431

7

Homologous desensitization with gonadotropin-releasing hormone (GnRH) also diminishes gonadotrope responsiveness to maitotoxin: a role for the GnRH receptor-regulated calcium ion channel in mediation of cellular desensitization.

Conn PM, Staley DD, Yasumoto T, Huckle WR, Janovick JA. Conn PM, et al. Mol Endocrinol. 1987 Feb;1(2):154-9. doi: 10.1210/mend-1-2-154. Mol Endocrinol. 1987. PMID: 2457798

8

Synthesis of gonadotropin-releasing hormone receptors by gonadotrope cell cultures: both preexisting receptors and those unmasked by protein kinase-C activators show a similar synthetic rate.

Braden TD, Hawes BE, Conn PM. Braden TD, et al. Among authors: conn pm. Endocrinology. 1989 Sep;125(3):1623-9. doi: 10.1210/endo-125-3-1623. Endocrinology. 1989. PMID: 2547592

9

Differential sensitivity of agonist- and antagonist-occupied gonadotropin-releasing hormone receptors to protein kinase C activators. A marker for receptor activation.

Huckle WR, McArdle CA, Conn PM. Huckle WR, et al. Among authors: conn pm. J Biol Chem. 1988 Mar 5;263(7):3296-302. J Biol Chem. 1988. PMID: 2830280 Free article.

10

Enhanced responsiveness of gonadotropes after protein kinase-C activation: postreceptor regulation of gonadotropin releasing hormone action.

McArdle CA, Huckle WR, Johnson LA, Conn PM. McArdle CA, et al. Among authors: conn pm. Endocrinology. 1988 May;122(5):1905-14. doi: 10.1210/endo-122-5-1905. Endocrinology. 1988. PMID: 2834179

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