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Multiple interaction nodes define the postreplication repair response to UV-induced DNA damage that is defective in melanomas and correlated with UV signature mutation load.
Pavey S, Pinder A, Fernando W, D'Arcy N, Matigian N, Skalamera D, Lê Cao KA, Loo-Oey D, Hill MM, Stark M, Kimlin M, Burgess A, Cloonan N, Sturm RA, Gabrielli B. Pavey S, et al. Among authors: gabrielli b. Mol Oncol. 2020 Jan;14(1):22-41. doi: 10.1002/1878-0261.12601. Epub 2019 Dec 19. Mol Oncol. 2020. PMID: 31733171 Free PMC article.
Endogenous Replication Stress Marks Melanomas Sensitive to CHEK1 Inhibitors In Vivo.
Oo ZY, Stevenson AJ, Proctor M, Daignault SM, Walpole S, Lanagan C, Chen J, Škalamera D, Spoerri L, Ainger SA, Sturm RA, Haass NK, Gabrielli B. Oo ZY, et al. Among authors: gabrielli b. Clin Cancer Res. 2018 Jun 15;24(12):2901-2912. doi: 10.1158/1078-0432.CCR-17-2701. Epub 2018 Mar 13. Clin Cancer Res. 2018. PMID: 29535131
Analyzing checkpoint controls in human skin.
Pavey S, Gabrielli BG. Pavey S, et al. Methods Mol Biol. 2004;280:175-83. doi: 10.1385/1-59259-788-2:175. Methods Mol Biol. 2004. PMID: 15187253
Decatenation checkpoint-defective melanomas are dependent on PI3K for survival.
Brooks K, Ranall M, Spoerri L, Stevenson A, Gunasingh G, Pavey S, Meunier F, Gonda TJ, Gabrielli B. Brooks K, et al. Among authors: gabrielli b. Pigment Cell Melanoma Res. 2014 Sep;27(5):813-21. doi: 10.1111/pcmr.12268. Epub 2014 Jun 23. Pigment Cell Melanoma Res. 2014. PMID: 24890688
148 results