Skoultchi AI - Search Results

1

A genetic screen and transcript profiling reveal a shared regulatory program for Drosophila linker histone H1 and chromatin remodeler CHD1.

Kavi H, Lu X, Xu N, Bartholdy BA, Vershilova E, Skoultchi AI, Fyodorov DV. Kavi H, et al. Among authors: skoultchi ai. G3 (Bethesda). 2015 Jan 27;5(4):677-87. doi: 10.1534/g3.115.016709. G3 (Bethesda). 2015. PMID: 25628309 Free PMC article.

2

Linker histone H1 is essential for Drosophila development, the establishment of pericentric heterochromatin, and a normal polytene chromosome structure.

Lu X, Wontakal SN, Emelyanov AV, Morcillo P, Konev AY, Fyodorov DV, Skoultchi AI. Lu X, et al. Among authors: skoultchi ai. Genes Dev. 2009 Feb 15;23(4):452-65. doi: 10.1101/gad.1749309. Epub 2009 Feb 4. Genes Dev. 2009. PMID: 19196654 Free PMC article.

3

Drosophila H1 regulates the genetic activity of heterochromatin by recruitment of Su(var)3-9.

Lu X, Wontakal SN, Kavi H, Kim BJ, Guzzardo PM, Emelyanov AV, Xu N, Hannon GJ, Zavadil J, Fyodorov DV, Skoultchi AI. Lu X, et al. Among authors: skoultchi ai. Science. 2013 Apr 5;340(6128):78-81. doi: 10.1126/science.1234654. Science. 2013. PMID: 23559249 Free PMC article.

4

Local compartment changes and regulatory landscape alterations in histone H1-depleted cells.

Geeven G, Zhu Y, Kim BJ, Bartholdy BA, Yang SM, Macfarlan TS, Gifford WD, Pfaff SL, Verstegen MJ, Pinto H, Vermunt MW, Creyghton MP, Wijchers PJ, Stamatoyannopoulos JA, Skoultchi AI, de Laat W. Geeven G, et al. Among authors: skoultchi ai. Genome Biol. 2015 Dec 23;16:289. doi: 10.1186/s13059-015-0857-0. Genome Biol. 2015. PMID: 26700097 Free PMC article.

5

Independent Biological and Biochemical Functions for Individual Structural Domains of Drosophila Linker Histone H1.

Kavi H, Emelyanov AV, Fyodorov DV, Skoultchi AI. Kavi H, et al. Among authors: skoultchi ai. J Biol Chem. 2016 Jul 15;291(29):15143-55. doi: 10.1074/jbc.M116.730705. Epub 2016 May 18. J Biol Chem. 2016. PMID: 27226620 Free PMC article.

6

BEN domain protein Elba2 can functionally substitute for linker histone H1 in Drosophila in vivo.

Xu N, Lu X, Kavi H, Emelyanov AV, Bernardo TJ, Vershilova E, Skoultchi AI, Fyodorov DV. Xu N, et al. Among authors: skoultchi ai. Sci Rep. 2016 Sep 30;6:34354. doi: 10.1038/srep34354. Sci Rep. 2016. PMID: 27687115 Free PMC article.

7

Regulatory functions and chromatin loading dynamics of linker histone H1 during endoreplication in Drosophila.

Andreyeva EN, Bernardo TJ, Kolesnikova TD, Lu X, Yarinich LA, Bartholdy BA, Guo X, Posukh OV, Healton S, Willcockson MA, Pindyurin AV, Zhimulev IF, Skoultchi AI, Fyodorov DV. Andreyeva EN, et al. Among authors: skoultchi ai. Genes Dev. 2017 Mar 15;31(6):603-616. doi: 10.1101/gad.295717.116. Genes Dev. 2017. PMID: 28404631 Free PMC article.

8

Emerging roles of linker histones in regulating chromatin structure and function.

Fyodorov DV, Zhou BR, Skoultchi AI, Bai Y. Fyodorov DV, et al. Among authors: skoultchi ai. Nat Rev Mol Cell Biol. 2018 Mar;19(3):192-206. doi: 10.1038/nrm.2017.94. Epub 2017 Oct 11. Nat Rev Mol Cell Biol. 2018. PMID: 29018282 Free PMC article. Review.

9

Drosophila linker histone H1 coordinates STAT-dependent organization of heterochromatin and suppresses tumorigenesis caused by hyperactive JAK-STAT signaling.

Xu N, Emelyanov AV, Fyodorov DV, Skoultchi AI. Xu N, et al. Among authors: skoultchi ai. Epigenetics Chromatin. 2014 Jul 28;7:16. doi: 10.1186/1756-8935-7-16. eCollection 2014. Epigenetics Chromatin. 2014. PMID: 25177369 Free PMC article.

10

H1 histones control the epigenetic landscape by local chromatin compaction.

Willcockson MA, Healton SE, Weiss CN, Bartholdy BA, Botbol Y, Mishra LN, Sidhwani DS, Wilson TJ, Pinto HB, Maron MI, Skalina KA, Toro LN, Zhao J, Lee CH, Hou H, Yusufova N, Meydan C, Osunsade A, David Y, Cesarman E, Melnick AM, Sidoli S, Garcia BA, Edelmann W, Macian F, Skoultchi AI. Willcockson MA, et al. Among authors: skoultchi ai. Nature. 2021 Jan;589(7841):293-298. doi: 10.1038/s41586-020-3032-z. Epub 2020 Dec 9. Nature. 2021. PMID: 33299182 Free PMC article.

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