Grimm M - Search Results

1

CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation.

Grimm M, Ling H, Willeford A, Pereira L, Gray CB, Erickson JR, Sarma S, Respress JL, Wehrens XH, Bers DM, Brown JH. Grimm M, et al. J Mol Cell Cardiol. 2015 Aug;85:282-91. doi: 10.1016/j.yjmcc.2015.06.007. Epub 2015 Jun 14. J Mol Cell Cardiol. 2015. PMID: 26080362 Free PMC article.

2

Beta-adrenergic receptor signaling in the heart: role of CaMKII.

Grimm M, Brown JH. Grimm M, et al. J Mol Cell Cardiol. 2010 Feb;48(2):322-30. doi: 10.1016/j.yjmcc.2009.10.016. Epub 2009 Oct 31. J Mol Cell Cardiol. 2010. PMID: 19883653 Free PMC article. Review.

3

Crossing signals: relationships between β-adrenergic stimulation and CaMKII activation.

Grimm M, Ling H, Brown JH. Grimm M, et al. Heart Rhythm. 2011 Aug;8(8):1296-8. doi: 10.1016/j.hrthm.2011.02.027. Epub 2011 Feb 23. Heart Rhythm. 2011. PMID: 21354330 Free PMC article. No abstract available.

4

beta-Adrenergic receptor stimulated Ncx1 upregulation is mediated via a CaMKII/AP-1 signaling pathway in adult cardiomyocytes.

Mani SK, Egan EA, Addy BK, Grimm M, Kasiganesan H, Thiyagarajan T, Renaud L, Brown JH, Kern CB, Menick DR. Mani SK, et al. Among authors: grimm m. J Mol Cell Cardiol. 2010 Feb;48(2):342-51. doi: 10.1016/j.yjmcc.2009.11.007. Epub 2009 Nov 27. J Mol Cell Cardiol. 2010. PMID: 19945464 Free PMC article.

5

Cardiac hypertrophy and heart failure development through Gq and CaM kinase II signaling.

Mishra S, Ling H, Grimm M, Zhang T, Bers DM, Brown JH. Mishra S, et al. Among authors: grimm m. J Cardiovasc Pharmacol. 2010 Dec;56(6):598-603. doi: 10.1097/FJC.0b013e3181e1d263. J Cardiovasc Pharmacol. 2010. PMID: 20531218 Free PMC article. Review.

6

Ca2+/Calmodulin-dependent protein kinase II δ mediates myocardial ischemia/reperfusion injury through nuclear factor-κB.

Ling H, Gray CB, Zambon AC, Grimm M, Gu Y, Dalton N, Purcell NH, Peterson K, Brown JH. Ling H, et al. Among authors: grimm m. Circ Res. 2013 Mar 15;112(6):935-44. doi: 10.1161/CIRCRESAHA.112.276915. Epub 2013 Feb 6. Circ Res. 2013. PMID: 23388157 Free PMC article.

7

Reduced contractile response to alpha1-adrenergic stimulation in atria from mice with chronic cardiac calmodulin kinase II inhibition.

Grimm M, El-Armouche A, Zhang R, Anderson ME, Eschenhagen T. Grimm M, et al. J Mol Cell Cardiol. 2007 Mar;42(3):643-52. doi: 10.1016/j.yjmcc.2006.12.010. Epub 2006 Dec 28. J Mol Cell Cardiol. 2007. PMID: 17292391

8

Inactivation of (Gialpha) proteins increases arrhythmogenic effects of beta-adrenergic stimulation in the heart.

Grimm M, Gsell S, Mittmann C, Nose M, Scholz H, Weil J, Eschenhagen T. Grimm M, et al. J Mol Cell Cardiol. 1998 Oct;30(10):1917-28. doi: 10.1006/jmcc.1998.0769. J Mol Cell Cardiol. 1998. PMID: 9799646

9

The MLCK-mediated alpha1-adrenergic inotropic effect in atrial myocardium is negatively modulated by PKCepsilon signaling.

Grimm M, Mahnecke N, Soja F, El-Armouche A, Haas P, Treede H, Reichenspurner H, Eschenhagen T. Grimm M, et al. Br J Pharmacol. 2006 Aug;148(7):991-1000. doi: 10.1038/sj.bjp.0706803. Epub 2006 Jun 19. Br J Pharmacol. 2006. PMID: 16783412 Free PMC article.

10

Phosphatase inhibitor-1-deficient mice are protected from catecholamine-induced arrhythmias and myocardial hypertrophy.

El-Armouche A, Wittköpper K, Degenhardt F, Weinberger F, Didié M, Melnychenko I, Grimm M, Peeck M, Zimmermann WH, Unsöld B, Hasenfuss G, Dobrev D, Eschenhagen T. El-Armouche A, et al. Among authors: grimm m. Cardiovasc Res. 2008 Dec 1;80(3):396-406. doi: 10.1093/cvr/cvn208. Epub 2008 Aug 8. Cardiovasc Res. 2008. PMID: 18689792

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