Quinn A - Search Results

1

High-throughput sequencing reveals restricted TCR Vβ usage and public TCRβ clonotypes among pancreatic lymph node memory CD4(+) T cells and their involvement in autoimmune diabetes.

Marrero I, Aguilera C, Hamm DE, Quinn A, Kumar V. Marrero I, et al. Among authors: quinn a. Mol Immunol. 2016 Jun;74:82-95. doi: 10.1016/j.molimm.2016.04.013. Epub 2016 May 6. Mol Immunol. 2016. PMID: 27161799 Free PMC article.

2

N-terminal flanking residues of a diabetes-associated GAD65 determinant are necessary for activation of antigen-specific T cells in diabetes-resistant mice.

Dai YD, Jensen KP, Marrero I, Li N, Quinn A, Sercarz EE. Dai YD, et al. Among authors: quinn a. Eur J Immunol. 2008 Apr;38(4):968-76. doi: 10.1002/eji.200737703. Eur J Immunol. 2008. PMID: 18395850 Free article.

3

Dominant CTL-inducing epitopes on GAD65 are adjacent to or overlap with dominant Th-inducing epitopes.

Busick RY, Aguilera C, Quinn A. Busick RY, et al. Among authors: quinn a. Clin Immunol. 2007 Mar;122(3):298-311. doi: 10.1016/j.clim.2006.10.008. Epub 2006 Dec 14. Clin Immunol. 2007. PMID: 17174605

4

Insulin-dependent diabetes loci Idd5 and Idd9 increase sensitivity to experimental autoimmune encephalomyelitis.

Mayo S, Kohler W, Kumar V, Quinn A. Mayo S, et al. Among authors: quinn a. Clin Immunol. 2006 Feb-Mar;118(2-3):219-28. doi: 10.1016/j.clim.2005.10.006. Epub 2005 Nov 21. Clin Immunol. 2006. PMID: 16301001

5

Interactions of effectors and regulators are decisive in the manifestations of type 1 diabetes in nonobese diabetic mice.

Quinn A, Kumar V, Jensen KP, Sercarz EE. Quinn A, et al. Curr Dir Autoimmun. 2001;4:171-92. doi: 10.1159/000060537. Curr Dir Autoimmun. 2001. PMID: 11569402 Review. No abstract available.

6

T cells to a dominant epitope of GAD65 express a public CDR3 motif.

Quinn A, McInerney M, Huffman D, McInerney B, Mayo S, Haskins K, Sercarz E. Quinn A, et al. Int Immunol. 2006 Jun;18(6):967-79. doi: 10.1093/intimm/dxl033. Epub 2006 Apr 26. Int Immunol. 2006. PMID: 16641112

7

Regulatory and effector CD4 T cells in nonobese diabetic mice recognize overlapping determinants on glutamic acid decarboxylase and use distinct V beta genes.

Quinn A, McInerney B, Reich EP, Kim O, Jensen KP, Sercarz EE. Quinn A, et al. J Immunol. 2001 Mar 1;166(5):2982-91. doi: 10.4049/jimmunol.166.5.2982. J Immunol. 2001. PMID: 11207247

8

Relative resistance to nasally induced tolerance in non-obese diabetic mice but not other I-A(g7)-expressing mouse strains.

Quinn A, Melo M, Ethell D, Sercarz EE. Quinn A, et al. Int Immunol. 2001 Oct;13(10):1321-33. doi: 10.1093/intimm/13.10.1321. Int Immunol. 2001. PMID: 11581177

9

MHC class I-restricted determinants on the glutamic acid decarboxylase 65 molecule induce spontaneous CTL activity.

Quinn A, McInerney MF, Sercarz EE. Quinn A, et al. J Immunol. 2001 Aug 1;167(3):1748-57. doi: 10.4049/jimmunol.167.3.1748. J Immunol. 2001. PMID: 11466400

10

Molecular profile of the T cell receptors of regulatory and effector CD4+ T cells recognizing overlapping determinants on glutamic acid decarboxylase (524-543).

Im JS, Quinn A, Sercarz EE, Lake DF. Im JS, et al. Among authors: quinn a. Mol Immunol. 2004 Feb;40(13):971-80. doi: 10.1016/j.molimm.2003.09.007. Mol Immunol. 2004. PMID: 14725793

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