Masliah E - Search Results

1

Amyloid Accumulation Drives Proteome-wide Alterations in Mouse Models of Alzheimer's Disease-like Pathology.

Savas JN, Wang YZ, DeNardo LA, Martinez-Bartolome S, McClatchy DB, Hark TJ, Shanks NF, Cozzolino KA, Lavallée-Adam M, Smukowski SN, Park SK, Kelly JW, Koo EH, Nakagawa T, Masliah E, Ghosh A, Yates JR 3rd. Savas JN, et al. Among authors: masliah e. Cell Rep. 2017 Nov 28;21(9):2614-2627. doi: 10.1016/j.celrep.2017.11.009. Cell Rep. 2017. PMID: 29186695 Free PMC article.

2

Amyloid beta protein toxicity mediated by the formation of amyloid-beta protein precursor complexes.

Lu DC, Shaked GM, Masliah E, Bredesen DE, Koo EH. Lu DC, et al. Among authors: masliah e. Ann Neurol. 2003 Dec;54(6):781-9. doi: 10.1002/ana.10761. Ann Neurol. 2003. PMID: 14681887

3

Axonopathy and transport deficits early in the pathogenesis of Alzheimer's disease.

Stokin GB, Lillo C, Falzone TL, Brusch RG, Rockenstein E, Mount SL, Raman R, Davies P, Masliah E, Williams DS, Goldstein LS. Stokin GB, et al. Among authors: masliah e. Science. 2005 Feb 25;307(5713):1282-8. doi: 10.1126/science.1105681. Science. 2005. PMID: 15731448

4

Perturbed neurogenesis in the adult hippocampus associated with presenilin-1 A246E mutation.

Chevallier NL, Soriano S, Kang DE, Masliah E, Hu G, Koo EH. Chevallier NL, et al. Among authors: masliah e. Am J Pathol. 2005 Jul;167(1):151-9. doi: 10.1016/S0002-9440(10)62962-8. Am J Pathol. 2005. PMID: 15972961 Free PMC article.

5

High beta-secretase activity elicits neurodegeneration in transgenic mice despite reductions in amyloid-beta levels: implications for the treatment of Alzheimer disease.

Rockenstein E, Mante M, Alford M, Adame A, Crews L, Hashimoto M, Esposito L, Mucke L, Masliah E. Rockenstein E, et al. Among authors: masliah e. J Biol Chem. 2005 Sep 23;280(38):32957-67. doi: 10.1074/jbc.M507016200. Epub 2005 Jul 15. J Biol Chem. 2005. PMID: 16027115 Free article.

6

Cerebrolysin decreases amyloid-beta production by regulating amyloid protein precursor maturation in a transgenic model of Alzheimer's disease.

Rockenstein E, Torrance M, Mante M, Adame A, Paulino A, Rose JB, Crews L, Moessler H, Masliah E. Rockenstein E, et al. Among authors: masliah e. J Neurosci Res. 2006 May 15;83(7):1252-61. doi: 10.1002/jnr.20818. J Neurosci Res. 2006. PMID: 16511867

7

Synaptic remodeling during aging and in Alzheimer's disease.

Masliah E, Crews L, Hansen L. Masliah E, et al. J Alzheimers Dis. 2006;9(3 Suppl):91-9. doi: 10.3233/jad-2006-9s311. J Alzheimers Dis. 2006. PMID: 16914848 Review.

8

Neuroprotective effects of regulators of the glycogen synthase kinase-3beta signaling pathway in a transgenic model of Alzheimer's disease are associated with reduced amyloid precursor protein phosphorylation.

Rockenstein E, Torrance M, Adame A, Mante M, Bar-on P, Rose JB, Crews L, Masliah E. Rockenstein E, et al. Among authors: masliah e. J Neurosci. 2007 Feb 21;27(8):1981-91. doi: 10.1523/JNEUROSCI.4321-06.2007. J Neurosci. 2007. PMID: 17314294 Free PMC article.

9

The autophagy-related protein beclin 1 shows reduced expression in early Alzheimer disease and regulates amyloid beta accumulation in mice.

Pickford F, Masliah E, Britschgi M, Lucin K, Narasimhan R, Jaeger PA, Small S, Spencer B, Rockenstein E, Levine B, Wyss-Coray T. Pickford F, et al. Among authors: masliah e. J Clin Invest. 2008 Jun;118(6):2190-9. doi: 10.1172/JCI33585. J Clin Invest. 2008. PMID: 18497889 Free PMC article.

10

Biological transgenic mouse models of Alzheimer's disease.

Crews L, Rockenstein E, Masliah E. Crews L, et al. Among authors: masliah e. Handb Clin Neurol. 2008;89:291-301. doi: 10.1016/S0072-9752(07)01227-4. Handb Clin Neurol. 2008. PMID: 18631753 Review. No abstract available.

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