Ostadal B - Search Results

1

Proteomic analysis of cardiac ventricles: baso-apical differences.

Eckhardt A, Kulhava L, Miksik I, Pataridis S, Hlavackova M, Vasinova J, Kolar F, Sedmera D, Ostadal B. Eckhardt A, et al. Among authors: ostadal b. Mol Cell Biochem. 2018 Aug;445(1-2):211-219. doi: 10.1007/s11010-017-3266-8. Epub 2018 Jan 4. Mol Cell Biochem. 2018. PMID: 29302836

2

Developmental determinants of cardiac sensitivity to hypoxia.

Ostadal B, Ostadalova I, Kolar F, Sedmera D. Ostadal B, et al. Can J Physiol Pharmacol. 2014 Jul;92(7):566-74. doi: 10.1139/cjpp-2013-0498. Epub 2014 Apr 3. Can J Physiol Pharmacol. 2014. PMID: 24873901 Review.

3

Cardiac phosphocreatine deficiency induced by GPA during postnatal development in rat.

Pelouch V, Kolár F, Khuchua ZA, Elizarova GV, Milerová M, Ost'ádal B, Saks VA. Pelouch V, et al. Mol Cell Biochem. 1996 Oct-Nov;163-164:67-76. doi: 10.1007/BF00408642. Mol Cell Biochem. 1996. PMID: 8974041

4

Effect of the preweaning nutritional state on the cardiac protein profile and functional performance of the rat heart.

Pelouch V, Kolár F, Milerová M, Ostádal B. Pelouch V, et al. Among authors: ostadal b. Mol Cell Biochem. 1997 Dec;177(1-2):221-8. doi: 10.1023/a:1006823608341. Mol Cell Biochem. 1997. PMID: 9450666

5

Tolerance to ischaemia and ischaemic preconditioning in neonatal rat heart.

Ostadalova I, Ostadal B, Kolár F, Parratt JR, Wilson S. Ostadalova I, et al. Among authors: ostadal b. J Mol Cell Cardiol. 1998 Apr;30(4):857-65. doi: 10.1006/jmcc.1998.0653. J Mol Cell Cardiol. 1998. PMID: 9602435

6

Developmental and sex differences in cardiac tolerance to ischemia-reperfusion injury: the role of mitochondria ¹.

Ostadal B, Drahota Z, Houstek J, Milerova M, Ostadalova I, Hlavackova M, Kolar F. Ostadal B, et al. Can J Physiol Pharmacol. 2019 Sep;97(9):808-814. doi: 10.1139/cjpp-2019-0060. Epub 2019 Mar 20. Can J Physiol Pharmacol. 2019. PMID: 30893574 Review.

7

Selective replacement of mitochondrial DNA increases the cardioprotective effect of chronic continuous hypoxia in spontaneously hypertensive rats.

Neckář J, Svatoňová A, Weissová R, Drahota Z, Zajíčková P, Brabcová I, Kolář D, Alánová P, Vašinová J, Šilhavý J, Hlaváčková M, Tauchmannová K, Milerová M, Ošťádal B, Červenka L, Žurmanová J, Kalous M, Nováková O, Novotný J, Pravenec M, Kolář F. Neckář J, et al. Among authors: ostadal b. Clin Sci (Lond). 2017 May 1;131(9):865-881. doi: 10.1042/CS20170083. Epub 2017 Mar 14. Clin Sci (Lond). 2017. PMID: 28292971

8

Left ventricular function and remodelling in rats exposed stepwise up to extreme chronic intermittent hypoxia.

Papoušek F, Sedmera D, Neckář J, Ošťádal B, Kolář F. Papoušek F, et al. Among authors: ostadal b. Respir Physiol Neurobiol. 2020 Nov;282:103526. doi: 10.1016/j.resp.2020.103526. Epub 2020 Aug 15. Respir Physiol Neurobiol. 2020. PMID: 32805421

9

Right ventricular function in rats with hypoxic pulmonary hypertension.

Kolár F, Ostádal B. Kolár F, et al. Among authors: ostadal b. Pflugers Arch. 1991 Sep;419(2):121-6. doi: 10.1007/BF00372996. Pflugers Arch. 1991. PMID: 1835788

10

Adaptation to high altitude hypoxia protects the rat heart against ischemia-induced arrhythmias. Involvement of mitochondrial K(ATP) channel.

Asemu G, Papousek F, Ostádal B, Kolár F. Asemu G, et al. Among authors: ostadal b. J Mol Cell Cardiol. 1999 Oct;31(10):1821-31. doi: 10.1006/jmcc.1999.1013. J Mol Cell Cardiol. 1999. PMID: 10525420

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