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A collective diabetes cross in combination with a computational framework to dissect the genetics of human obesity and Type 2 diabetes.
Vogel H, Kamitz A, Hallahan N, Lebek S, Schallschmidt T, Jonas W, Jähnert M, Gottmann P, Zellner L, Kanzleiter T, Damen M, Altenhofen D, Burkhardt R, Renner S, Dahlhoff M, Wolf E, Müller TD, Blüher M, Joost HG, Chadt A, Al-Hasani H, Schürmann A. Vogel H, et al. Among authors: schurmann a. Hum Mol Genet. 2018 Sep 1;27(17):3099-3112. doi: 10.1093/hmg/ddy217. Hum Mol Genet. 2018. PMID: 29893858 Free PMC article.
Subcellular fractionation of adipocytes and 3T3-L1 cells.
Joost HG, Schürmann A. Joost HG, et al. Among authors: schurmann a. Methods Mol Biol. 2001;155:77-82. doi: 10.1385/1-59259-231-7:077. Methods Mol Biol. 2001. PMID: 11293085 Review. No abstract available.
Targeting of GLUT6 (formerly GLUT9) and GLUT8 in rat adipose cells.
Lisinski I, Schürmann A, Joost HG, Cushman SW, Al-Hasani H. Lisinski I, et al. Among authors: schurmann a. Biochem J. 2001 Sep 1;358(Pt 2):517-22. doi: 10.1042/0264-6021:3580517. Biochem J. 2001. PMID: 11513753 Free PMC article.
Likewise, when endocytosis was blocked by co-expression of a dominant-negative mutant of the dynamin GTPase, wild-type HA-GLUT6 and HA-GLUT8 accumulated on the cell surface. ...Thus GLUT6 and GLUT8 appear to recycle in a dynamin-dependent manner between internal mem …
Likewise, when endocytosis was blocked by co-expression of a dominant-negative mutant of the dynamin GTPase, wild-type HA-GLUT6 and H …
278 results