Jaggi R - Search Results

1

Induction of transforming growth factor alpha expression in mouse mammary epithelial cells after transformation with a point-mutated c-Ha-ras protooncogene.

Ciardiello F, Kim N, Hynes N, Jaggi R, Redmond S, Liscia DS, Sanfilippo B, Merlo G, Callahan R, Kidwell WR, et al. Ciardiello F, et al. Among authors: jaggi r. Mol Endocrinol. 1988 Dec;2(12):1202-16. doi: 10.1210/mend-2-12-1202. Mol Endocrinol. 1988. PMID: 3063955

2

New acceptor cell for transfected genomic DNA: oncogene transfer into a mouse mammary epithelial cell line.

Hynes NE, Jaggi R, Kozma SC, Ball R, Muellener D, Wetherall NT, Davis BW, Groner B. Hynes NE, et al. Among authors: jaggi r. Mol Cell Biol. 1985 Jan;5(1):268-72. doi: 10.1128/mcb.5.1.268-272.1985. Mol Cell Biol. 1985. PMID: 3982419 Free PMC article.

3

The v-mos and c-Ha-ras oncoproteins exert similar effects on the pattern of protein synthesis.

Klemenz R, Hoffmann S, Jaggi R, Werenskiold AK. Klemenz R, et al. Among authors: jaggi r. Oncogene. 1989 Jun;4(6):799-803. Oncogene. 1989. PMID: 2525241

4

Overexpression of Mos, Ras, Src, and Fos inhibits mouse mammary epithelial cell differentiation.

Jehn B, Costello E, Marti A, Keon N, Deane R, Li F, Friis RR, Burri PH, Martin F, Jaggi R. Jehn B, et al. Among authors: jaggi r. Mol Cell Biol. 1992 Sep;12(9):3890-902. doi: 10.1128/mcb.12.9.3890-3902.1992. Mol Cell Biol. 1992. PMID: 1508191 Free PMC article.

5

Desensitization of the Ca2+-mobilizing system to serum growth factors by Ha-ras and v-mos.

Maly K, Doppler W, Oberhuber H, Meusburger H, Hofmann J, Jaggi R, Grunicke HH. Maly K, et al. Among authors: jaggi r. Mol Cell Biol. 1988 Oct;8(10):4212-6. doi: 10.1128/mcb.8.10.4212-4216.1988. Mol Cell Biol. 1988. PMID: 3141785 Free PMC article.

In a preceding report it was demonstrated that the expression of Ha-ras or v-mos in cells transfected with Ha-ras or v-mos, respectively, leads to an activation of the Na+/H+ antiporter and a concomitant rise in intracellular pH (W. Doppler, R. Jaggi, and B. Groner, …

In a preceding report it was demonstrated that the expression of Ha-ras or v-mos in cells transfected with Ha-ras or v-mos, respectively, le …

6

Induction of v-mos and activated Ha-ras oncogene expression in quiescent NIH 3T3 cells causes intracellular alkalinisation and cell-cycle progression.

Doppler W, Jaggi R, Groner B. Doppler W, et al. Among authors: jaggi r. Gene. 1987;54(1):147-53. doi: 10.1016/0378-1119(87)90357-x. Gene. 1987. PMID: 3038697

7

The v-mos and H-ras oncogene expression represses glucocorticoid hormone-dependent transcription from the mouse mammary tumor virus LTR.

Jaggi R, Salmons B, Muellener D, Groner B. Jaggi R, et al. EMBO J. 1986 Oct;5(10):2609-16. doi: 10.1002/j.1460-2075.1986.tb04541.x. EMBO J. 1986. PMID: 3023051 Free PMC article.

8

Oncogenes modulate cellular gene expression and repress glucocorticoid regulated gene transcription.

Jaggi R, Friis R, Groner B. Jaggi R, et al. J Steroid Biochem. 1988 May;29(5):457-63. doi: 10.1016/0022-4731(88)90179-3. J Steroid Biochem. 1988. PMID: 3379954

9

mos-induced inhibition of glucocorticoid receptor function is mediated by Fos.

Touray M, Ryan F, Saurer S, Martin F, Jaggi R. Touray M, et al. Among authors: jaggi r. Oncogene. 1991 Feb;6(2):211-7. Oncogene. 1991. PMID: 1847998

10

Characterisation of functional inhibition of the glucocorticoid receptor by Fos/Jun.

Touray M, Ryan F, Jaggi R, Martin F. Touray M, et al. Among authors: jaggi r. Oncogene. 1991 Jul;6(7):1227-34. Oncogene. 1991. PMID: 1650443

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