MacFarlane M - Search Results

1

Proteasome inhibition triggers the formation of TRAIL receptor 2 platforms for caspase-8 activation that accumulate in the cytosol.

Hellwig CT, Delgado ME, Skoko J, Dyck L, Hanna C, Wentges A, Langlais C, Hagenlocher C, Mack A, Dinsdale D, Cain K, MacFarlane M, Rehm M. Hellwig CT, et al. Among authors: macfarlane m. Cell Death Differ. 2022 Jan;29(1):147-155. doi: 10.1038/s41418-021-00843-7. Epub 2021 Aug 5. Cell Death Differ. 2022. PMID: 34354257 Free PMC article.

2

Formation of large molecular weight fragments of DNA is a key committed step of apoptosis in thymocytes.

Cohen GM, Sun XM, Fearnhead H, MacFarlane M, Brown DG, Snowden RT, Dinsdale D. Cohen GM, et al. Among authors: macfarlane m. J Immunol. 1994 Jul 15;153(2):507-16. J Immunol. 1994. PMID: 8021492

3

DNA degradation and proteolysis in thymocyte apoptosis.

Fearnhead HO, MacFarlane M, Dinsdale D, Cohen GM. Fearnhead HO, et al. Among authors: macfarlane m. Toxicol Lett. 1995 Dec;82-83:135-41. doi: 10.1016/0378-4274(95)03473-0. Toxicol Lett. 1995. PMID: 8597041

4

Processing/activation of at least four interleukin-1beta converting enzyme-like proteases occurs during the execution phase of apoptosis in human monocytic tumor cells.

MacFarlane M, Cain K, Sun XM, Alnemri ES, Cohen GM. MacFarlane M, et al. J Cell Biol. 1997 Apr 21;137(2):469-79. doi: 10.1083/jcb.137.2.469. J Cell Biol. 1997. PMID: 9128256 Free PMC article.

5

Active caspases and cleaved cytokeratins are sequestered into cytoplasmic inclusions in TRAIL-induced apoptosis.

MacFarlane M, Merrison W, Dinsdale D, Cohen GM. MacFarlane M, et al. J Cell Biol. 2000 Mar 20;148(6):1239-54. doi: 10.1083/jcb.148.6.1239. J Cell Biol. 2000. PMID: 10725337 Free PMC article.

6

Protein complexes activate distinct caspase cascades in death receptor and stress-induced apoptosis.

Bratton SB, MacFarlane M, Cain K, Cohen GM. Bratton SB, et al. Among authors: macfarlane m. Exp Cell Res. 2000 Apr 10;256(1):27-33. doi: 10.1006/excr.2000.4835. Exp Cell Res. 2000. PMID: 10739648 Review.

7

JNK (c-Jun N-terminal kinase) and p38 activation in receptor-mediated and chemically-induced apoptosis of T-cells: differential requirements for caspase activation.

MacFarlane M, Cohen GM, Dickens M. MacFarlane M, et al. Biochem J. 2000 May 15;348 Pt 1(Pt 1):93-101. Biochem J. 2000. PMID: 10794718 Free PMC article.

8

Macrophage-mediated clearance of cells undergoing caspase-3-independent death.

Turner C, Devitt A, Parker K, MacFarlane M, Giuliano M, Cohen GM, Gregory CD. Turner C, et al. Among authors: macfarlane m. Cell Death Differ. 2003 Mar;10(3):302-12. doi: 10.1038/sj.cdd.4401170. Cell Death Differ. 2003. PMID: 12700630

9

Pro-apoptotic proteins released from the mitochondria regulate the protein composition and caspase-processing activity of the native Apaf-1/caspase-9 apoptosome complex.

Twiddy D, Brown DG, Adrain C, Jukes R, Martin SJ, Cohen GM, MacFarlane M, Cain K. Twiddy D, et al. Among authors: macfarlane m. J Biol Chem. 2004 May 7;279(19):19665-82. doi: 10.1074/jbc.M311388200. Epub 2004 Mar 1. J Biol Chem. 2004. PMID: 14993223 Free article.

10

Caspase activation inhibits proteasome function during apoptosis.

Sun XM, Butterworth M, MacFarlane M, Dubiel W, Ciechanover A, Cohen GM. Sun XM, et al. Among authors: macfarlane m. Mol Cell. 2004 Apr 9;14(1):81-93. doi: 10.1016/s1097-2765(04)00156-x. Mol Cell. 2004. PMID: 15068805 Free article.

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