Pasha MAQ - Search Results

1

Susceptibility of CTLA-4 -1661A/G polymorphism towards severity of rheumatic heart disease.

Bansal A, Tasnim S, Gupta MD, Mp G, Batra V, Kohli S, Tyagi S, Pasha MAQ. Bansal A, et al. Among authors: pasha maq. Indian Heart J. 2021 Jul-Aug;73(4):511-515. doi: 10.1016/j.ihj.2021.05.002. Epub 2021 Jun 8. Indian Heart J. 2021. PMID: 34474769 Free PMC article.

2

Endothelial nitric oxide synthase gene haplotypes and circulating nitric oxide levels significantly associate with risk of essential hypertension.

Nejatizadeh A, Kumar R, Stobdan T, Goyal AK, Sikdar S, Gupta M, Javed S, Pasha MA. Nejatizadeh A, et al. Free Radic Biol Med. 2008 Jun 1;44(11):1912-8. doi: 10.1016/j.freeradbiomed.2008.02.004. Epub 2008 Feb 20. Free Radic Biol Med. 2008. PMID: 18325347

3

CYP11B2 gene haplotypes independently and in concurrence with aldosterone and aldosterone to renin ratio increase the risk of hypertension.

Nejatizadeh A, Kumar R, Stobdan T, Goyal AK, Gupta M, Tyagi S, Jain SK, Pasha MA. Nejatizadeh A, et al. Clin Biochem. 2010 Jan;43(1-2):136-41. doi: 10.1016/j.clinbiochem.2009.09.015. Epub 2009 Sep 26. Clin Biochem. 2010. PMID: 19786005

4

The epistasis between vascular homeostasis genes is apparent in essential hypertension.

Kumar R, Nejatizadeh A, Gupta M, Markan A, Tyagi S, Jain SK, Pasha MA. Kumar R, et al. Atherosclerosis. 2012 Feb;220(2):418-24. doi: 10.1016/j.atherosclerosis.2011.10.036. Epub 2011 Nov 2. Atherosclerosis. 2012. PMID: 22134143

5

Interactions between the FTO and GNB3 genes contribute to varied clinical phenotypes in hypertension.

Kumar R, Kohli S, Alam P, Barkotoky R, Gupta M, Tyagi S, Jain SK, Pasha MA. Kumar R, et al. PLoS One. 2013 May 14;8(5):e63934. doi: 10.1371/journal.pone.0063934. Print 2013. PLoS One. 2013. PMID: 23691120 Free PMC article.

6

Interactions between the genes of vasodilatation pathways influence blood pressure and nitric oxide level in hypertension.

Kumar R, Kohli S, Mishra A, Garg R, Alam P, Stobdan T, Nejatizadeh A, Gupta M, Tyagi S, Pasha MA. Kumar R, et al. Am J Hypertens. 2015 Feb;28(2):239-47. doi: 10.1093/ajh/hpu130. Epub 2014 Aug 26. Am J Hypertens. 2015. PMID: 25159081

7

Genetic differences and aberrant methylation in the apelin system predict the risk of high-altitude pulmonary edema.

Mishra A, Kohli S, Dua S, Thinlas T, Mohammad G, Pasha MA. Mishra A, et al. Proc Natl Acad Sci U S A. 2015 May 12;112(19):6134-9. doi: 10.1073/pnas.1422759112. Epub 2015 Apr 27. Proc Natl Acad Sci U S A. 2015. PMID: 25918383 Free PMC article.

8

Impact of interactions between risk alleles on clinical endpoints in hypertension.

Kohli S, Kumar R, Gupta M, Tyagi S, Pasha MA. Kohli S, et al. Heart Asia. 2016 May 19;8(1):83-9. doi: 10.1136/heartasia-2016-010723. eCollection 2016. Heart Asia. 2016. PMID: 27326240 Free PMC article.

9

The deleterious impact of a non-synonymous SNP on protein structure and function is apparent in hypertension.

Sharma K, Hema K, Bhatraju NK, Kukreti R, Das RS, Gupta MD, Syed MA, Pasha MAQ. Sharma K, et al. Among authors: pasha maq. J Mol Model. 2021 Dec 27;28(1):14. doi: 10.1007/s00894-021-04997-6. J Mol Model. 2021. PMID: 34961886

10

Association of CYP2E1 and NAT2 gene polymorphisms with chronic obstructive pulmonary disease.

Arif E, Vibhuti A, Alam P, Deepak D, Singh B, Athar M, Pasha MA. Arif E, et al. Clin Chim Acta. 2007 Jul;382(1-2):37-42. doi: 10.1016/j.cca.2007.03.013. Epub 2007 Mar 24. Clin Chim Acta. 2007. PMID: 17442289

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