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Author Correction: Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction in tafazzin-deficient cells.
Liang Z, Ralph-Epps T, Schmidtke MW, Lazcano P, Denis SW, Balážová M, Teixeira da Rosa N Jr, Chakkour M, Hazime S, Ren M, Schlame M, Houtkooper RH, Greenberg ML. Liang Z, et al. Among authors: balazova m. Sci Rep. 2024 Jun 25;14(1):14633. doi: 10.1038/s41598-024-65477-4. Sci Rep. 2024. PMID: 38918568 Free PMC article. No abstract available.
Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction in tafazzin-deficient cells.
Liang Z, Ralph-Epps T, Schmidtke MW, Lazcano P, Denis SW, Balážová M, Teixeira da Rosa N Jr, Chakkour M, Hazime S, Ren M, Schlame M, Houtkooper RH, Greenberg ML. Liang Z, et al. Among authors: balazova m. Sci Rep. 2024 May 20;14(1):11497. doi: 10.1038/s41598-024-62262-1. Sci Rep. 2024. PMID: 38769106 Free PMC article.
Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction and leads to increased glucose uptake in tafazzin-deficient cells.
Liang Z, Ralph-Epps T, Schmidtke MW, Lazcano P, Denis SW, Balážová M, Chakkour M, Hazime S, Ren M, Schlame M, Houtkooper RH, Greenberg ML. Liang Z, et al. Among authors: balazova m. bioRxiv [Preprint]. 2024 Feb 4:2024.02.03.578755. doi: 10.1101/2024.02.03.578755. bioRxiv. 2024. Update in: Sci Rep. 2024 May 20;14(1):11497. doi: 10.1038/s41598-024-62262-1 PMID: 38352304 Free PMC article. Updated. Preprint.
MyomiRs in cultured muscle cells from patients with idiopathic inflammatory myopathy are modulated by disease but not by 6-month exercise training.
Alchus Laiferová N, Nemec M, Vernerová L, Balážová M, Vokurková M, Oreská S, Klein M, Špiritović M, Tomčík M, Vencovský J, Ukropec J, Ukropcová B. Alchus Laiferová N, et al. Among authors: balazova m. Clin Exp Rheumatol. 2022 Feb;40(2):346-357. doi: 10.55563/clinexprheumatol/69z7za. Epub 2022 Feb 25. Clin Exp Rheumatol. 2022. PMID: 35225219 Free article.
Blocking phosphatidylglycerol degradation in yeast defective in cardiolipin remodeling results in a new model of the Barth syndrome cellular phenotype.
Káňovičová P, Čermáková P, Kubalová D, Bábelová L, Veselá P, Valachovič M, Zahumenský J, Horváth A, Malínský J, Balážová M. Káňovičová P, et al. Among authors: balazova m. J Biol Chem. 2022 Jan;298(1):101462. doi: 10.1016/j.jbc.2021.101462. Epub 2021 Dec 2. J Biol Chem. 2022. PMID: 34864056 Free PMC article.
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