Lin J - Search Results

1

Structure-destabilizing mutations unleash an intrinsic perforation activity of antiapoptotic Bcl-2 in the mitochondrial membrane enabling apoptotic cell death.

Gao P, Zhang Z, Wang R, Huang L, Wu H, Qiao Z, Wang X, Jin H, Peng J, Liu L, Chen Q, Lin J. Gao P, et al. Among authors: lin j. Mitochondrial Commun. 2023;1:48-61. doi: 10.1016/j.mitoco.2023.08.001. Epub 2023 Aug 9. Mitochondrial Commun. 2023. PMID: 39239250 Free PMC article.

2

The cytosolic domain of Bcl-2 forms small pores in model mitochondrial outer membrane after acidic pH-induced membrane association.

Peng J, Lapolla SM, Zhang Z, Lin J. Peng J, et al. Among authors: lin j. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi. 2009 Feb;26(1):130-7. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi. 2009. PMID: 19334571 Free PMC article.

3

The cytosolic domain of Bcl-2 oligomerizes to form pores in model mitochondrial outer membrane at acidic pH.

Peng J, Lapolla SM, Zhang Z, Lin J. Peng J, et al. Among authors: lin j. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi. 2009 Jun;26(3):631-7. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi. 2009. PMID: 19634687 Free PMC article.

4

The Bax BH3 peptide H2-H3 promotes apoptosis by inhibiting Bcl-2's pore-forming and anti-Bax activities in the membrane.

Peng J, Lapolla SM, Zhang Z, Lin J. Peng J, et al. Among authors: lin j. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi. 2009 Aug;26(4):829-35. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi. 2009. PMID: 19813621 Free PMC article.

5

Natural diterpenoid compound elevates expression of Bim protein, which interacts with antiapoptotic protein Bcl-2, converting it to proapoptotic Bax-like molecule.

Zhao L, He F, Liu H, Zhu Y, Tian W, Gao P, He H, Yue W, Lei X, Ni B, Wang X, Jin H, Hao X, Lin J, Chen Q. Zhao L, et al. Among authors: lin j. J Biol Chem. 2012 Jan 6;287(2):1054-65. doi: 10.1074/jbc.M111.264481. Epub 2011 Nov 7. J Biol Chem. 2012. PMID: 22065578 Free PMC article.

6

Photocrosslinking Approach to Investigate Protein Interactions in the BCL-2 Family.

Lin J, Johnson AE, Zhang Z. Lin J, et al. Methods Mol Biol. 2019;1877:131-149. doi: 10.1007/978-1-4939-8861-7_9. Methods Mol Biol. 2019. PMID: 30536003 Free PMC article.

7

Auto-activation of the apoptosis protein Bax increases mitochondrial membrane permeability and is inhibited by Bcl-2.

Tan C, Dlugosz PJ, Peng J, Zhang Z, Lapolla SM, Plafker SM, Andrews DW, Lin J. Tan C, et al. Among authors: lin j. J Biol Chem. 2006 May 26;281(21):14764-75. doi: 10.1074/jbc.M602374200. Epub 2006 Mar 29. J Biol Chem. 2006. PMID: 16571718 Free PMC article.

8

Oligomerization of membrane-bound Bcl-2 is involved in its pore formation induced by tBid.

Peng J, Ding J, Tan C, Baggenstoss B, Zhang Z, Lapolla SM, Lin J. Peng J, et al. Among authors: lin j. Apoptosis. 2009 Oct;14(10):1145-53. doi: 10.1007/s10495-009-0389-8. Apoptosis. 2009. PMID: 19701793 Free PMC article.

9

Dynamic PGAM5 multimers dephosphorylate BCL-xL or FUNDC1 to regulate mitochondrial and cellular fate.

Ma K, Zhang Z, Chang R, Cheng H, Mu C, Zhao T, Chen L, Zhang C, Luo Q, Lin J, Zhu Y, Chen Q. Ma K, et al. Among authors: lin j. Cell Death Differ. 2020 Mar;27(3):1036-1051. doi: 10.1038/s41418-019-0396-4. Epub 2019 Jul 31. Cell Death Differ. 2020. PMID: 31367011 Free PMC article.

10

Cysteine-based crosslinking approach for characterization of oligomeric pore-forming proteins in the mitochondrial membranes.

Zhang Z, Huang B, Zhang XC, Lin J. Zhang Z, et al. Among authors: lin j. Methods Enzymol. 2021;649:371-396. doi: 10.1016/bs.mie.2021.01.023. Epub 2021 Feb 12. Methods Enzymol. 2021. PMID: 33712193 Free PMC article.

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