Liu L - Search Results

1

Brucella induced upregulation of NO promote macrophages glycolysis through the NF-κB/G6PD pathway.

Cao S, Guo J, Zhu D, Sun Z, Liu L, Zhang Y, Maratbek S, Wang Z, Zhang J, Li W, Ding J, Deng X, Zhang H. Cao S, et al. Among authors: liu l. Int Immunopharmacol. 2024 Dec 5;142(Pt A):113038. doi: 10.1016/j.intimp.2024.113038. Epub 2024 Sep 13. Int Immunopharmacol. 2024. PMID: 39276450 Free article.

2

Deletion of the Type IV Secretion System Effector VceA Promotes Autophagy and Inhibits Apoptosis in Brucella-Infected Human Trophoblast Cells.

Zhang J, Li M, Li Z, Shi J, Zhang Y, Deng X, Liu L, Wang Z, Qi Y, Zhang H. Zhang J, et al. Among authors: liu l. Curr Microbiol. 2019 Apr;76(4):510-519. doi: 10.1007/s00284-019-01651-6. Epub 2019 Feb 25. Curr Microbiol. 2019. PMID: 30805699

3

Functional analysis of Bucella reveals transcriptional regulation of MarR.

Deng X, Li M, Liu L, Zhang J, Zhang Y, Guo J, Zhao T, Cao S, Li Z, Zhang H. Deng X, et al. Among authors: liu l. Microb Pathog. 2020 Jul;144:104201. doi: 10.1016/j.micpath.2020.104201. Epub 2020 Apr 20. Microb Pathog. 2020. PMID: 32325238

4

Survival characteristics and transcriptome profiling reveal the adaptive response of the Brucella melitensis 16M biofilm to osmotic stress.

Guo J, Zhu J, Zhao T, Sun Z, Song S, Zhang Y, Zhu D, Cao S, Deng X, Chai Y, Sun Y, Maratbek S, Chen C, Liu L, Zhang H. Guo J, et al. Among authors: liu l. Front Microbiol. 2022 Aug 17;13:968592. doi: 10.3389/fmicb.2022.968592. eCollection 2022. Front Microbiol. 2022. PMID: 36060772 Free PMC article.

5

The Flagellar Transcriptional Regulator FtcR Controls Brucella melitensis 16M Biofilm Formation via a betI-Mediated Pathway in Response to Hyperosmotic Stress.

Guo J, Deng X, Zhang Y, Song S, Zhao T, Zhu D, Cao S, Baryshnikov PI, Cao G, Blair HT, Chen C, Gu X, Liu L, Zhang H. Guo J, et al. Among authors: liu l. Int J Mol Sci. 2022 Aug 31;23(17):9905. doi: 10.3390/ijms23179905. Int J Mol Sci. 2022. PMID: 36077302 Free PMC article.

6

Brucella abortus modulates macrophage polarization and inflammatory response by targeting glutaminases through the NF-κB signaling pathway.

Zhao T, Zhang Z, Li Y, Sun Z, Liu L, Deng X, Guo J, Zhu D, Cao S, Chai Y, Nikolaevna UV, Maratbek S, Wang Z, Zhang H. Zhao T, et al. Among authors: liu l. Front Immunol. 2023 May 31;14:1180837. doi: 10.3389/fimmu.2023.1180837. eCollection 2023. Front Immunol. 2023. PMID: 37325614 Free PMC article.

7

Systemic Pharmacology Reveals the Potential Targets and Signaling Mechanisms in the Adjuvant Treatment of Brucellosis with Traditional Chinese Medicine.

Zhao T, Zhang Y, Liu L, Deng X, Guo J, Cao S, Zhu D, Xu J, Nikolaevna UV, Maratbek S, Wang Z, Sun Z, Gu X, Zhang H. Zhao T, et al. Among authors: liu l. ACS Omega. 2023 Jul 28;8(31):28797-28812. doi: 10.1021/acsomega.3c03716. eCollection 2023 Aug 8. ACS Omega. 2023. PMID: 37576692 Free PMC article.

8

Fungi, including Pithomyces chartarum cause facial eczema and inflammation in grazing sheep in Western China.

Liu L, Zhang J, Qu W, Wang Y, Yan G, Li Z, Zhang Y, Zhang H. Liu L, et al. Microb Pathog. 2023 Dec;185:106451. doi: 10.1016/j.micpath.2023.106451. Epub 2023 Nov 20. Microb Pathog. 2023. PMID: 37981078

9

Brucella secretory protein VceA promotes FOXO1 entry into the nucleus to shift host cell metabolism toward glycolysis.

Cao S, Han X, Deng X, Guo J, Liu L, Zhang Y, Suleimenov M, Zhao T, Li W, Ding J, Xie S, Zhang H. Cao S, et al. Among authors: liu l. Acta Biochim Biophys Sin (Shanghai). 2024 Nov 22. doi: 10.3724/abbs.2024203. Online ahead of print. Acta Biochim Biophys Sin (Shanghai). 2024. PMID: 39574164 Free article.

10

Brucella abortus transcriptional regulator ArsR6 inhibits host pyroptosis via BAB_RS28760 by triggering the endoplasmic reticulum stress pathway.

Zhu D, Guo J, Deng X, Li M, Wang Y, Wang Z, Sun Z, Cao S, Zhao T, Xu Y, Liu L, Zhang H. Zhu D, et al. Among authors: liu l. Int Immunopharmacol. 2025 Jan 8;147:114001. doi: 10.1016/j.intimp.2024.114001. Online ahead of print. Int Immunopharmacol. 2025. PMID: 39787758 Free article.

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