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Correction: Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology.
Verma A, Shteinfer-Kuzmine A, Kamenetsky N, Pittala S, Paul A, Nahon Crystal E, Ouro A, Chalifa-Caspi V, Pandey SK, Monsonego A, Vardi N, Knafo S, Shoshan-Barmatz V. Verma A, et al. Among authors: pittala s. Transl Neurodegener. 2023 Jan 11;12(1):2. doi: 10.1186/s40035-023-00335-3. Transl Neurodegener. 2023. PMID: 36627723 Free PMC article. No abstract available.
Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology.
Verma A, Shteinfer-Kuzmine A, Kamenetsky N, Pittala S, Paul A, Nahon Crystal E, Ouro A, Chalifa-Caspi V, Pandey SK, Monsonego A, Vardi N, Knafo S, Shoshan-Barmatz V. Verma A, et al. Among authors: pittala s. Transl Neurodegener. 2022 Dec 28;11(1):58. doi: 10.1186/s40035-022-00329-7. Transl Neurodegener. 2022. PMID: 36578022 Free PMC article.
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