Kravtsova-Ivantsiv Y - Search Results

1

CXCL12 restricts tumor growth by suppressing the Ras, ERK1/2, c-Myc, and the immune checkpoint PD-L1 pathways.

Kravtsova-Ivantsiv Y, Goldhirsh G, Ciechanover A. Kravtsova-Ivantsiv Y, et al. Proc Natl Acad Sci U S A. 2024 Dec 24;121(52):e2416909121. doi: 10.1073/pnas.2416909121. Epub 2024 Dec 17. Proc Natl Acad Sci U S A. 2024. PMID: 39689179

2

The N-terminal domain of MyoD is necessary and sufficient for its nuclear localization-dependent degradation by the ubiquitin system.

Sadeh R, Breitschopf K, Bercovich B, Zoabi M, Kravtsova-Ivantsiv Y, Kornitzer D, Schwartz A, Ciechanover A. Sadeh R, et al. Proc Natl Acad Sci U S A. 2008 Oct 14;105(41):15690-5. doi: 10.1073/pnas.0808373105. Epub 2008 Oct 3. Proc Natl Acad Sci U S A. 2008. PMID: 18836078 Free PMC article.

3

Modification by single ubiquitin moieties rather than polyubiquitination is sufficient for proteasomal processing of the p105 NF-kappaB precursor.

Kravtsova-Ivantsiv Y, Cohen S, Ciechanover A. Kravtsova-Ivantsiv Y, et al. Mol Cell. 2009 Feb 27;33(4):496-504. doi: 10.1016/j.molcel.2009.01.023. Mol Cell. 2009. PMID: 19250910 Free article.

4

ABIN-1 negatively regulates NF-kappaB by inhibiting processing of the p105 precursor.

Cohen S, Ciechanover A, Kravtsova-Ivantsiv Y, Lapid D, Lahav-Baratz S. Cohen S, et al. Biochem Biophys Res Commun. 2009 Nov 13;389(2):205-10. doi: 10.1016/j.bbrc.2009.08.074. Epub 2009 Aug 18. Biochem Biophys Res Commun. 2009. PMID: 19695220

5

Modification by single ubiquitin moieties rather than polyubiquitination is sufficient for proteasomal processing of the p105 NF-κB precursor.

Kravtsova-Ivantsiv Y, Cohen S, Ciechanover A. Kravtsova-Ivantsiv Y, et al. Adv Exp Med Biol. 2011;691:95-106. doi: 10.1007/978-1-4419-6612-4_10. Adv Exp Med Biol. 2011. PMID: 21153313 No abstract available.

6

Ubiquitination and degradation of proteins.

Kravtsova-Ivantsiv Y, Ciechanover A. Kravtsova-Ivantsiv Y, et al. Methods Mol Biol. 2011;753:335-57. doi: 10.1007/978-1-61779-148-2_23. Methods Mol Biol. 2011. PMID: 21604134

7

Non-canonical ubiquitin-based signals for proteasomal degradation.

Kravtsova-Ivantsiv Y, Ciechanover A. Kravtsova-Ivantsiv Y, et al. J Cell Sci. 2012 Feb 1;125(Pt 3):539-48. doi: 10.1242/jcs.093567. J Cell Sci. 2012. PMID: 22389393 Review.

8

Generation of free ubiquitin chains is up-regulated in stress and facilitated by the HECT domain ubiquitin ligases UFD4 and HUL5.

Braten O, Shabek N, Kravtsova-Ivantsiv Y, Ciechanover A. Braten O, et al. Biochem J. 2012 Jun 15;444(3):611-7. doi: 10.1042/BJ20111840. Biochem J. 2012. PMID: 22497224

9

The lysine48-based polyubiquitin chain proteasomal signal: not a single child anymore.

Kravtsova-Ivantsiv Y, Sommer T, Ciechanover A. Kravtsova-Ivantsiv Y, et al. Angew Chem Int Ed Engl. 2013 Jan 2;52(1):192-8. doi: 10.1002/anie.201205656. Epub 2012 Nov 4. Angew Chem Int Ed Engl. 2013. PMID: 23124625 Review.

10

Multiple sclerosis autoantigen myelin basic protein escapes control by ubiquitination during proteasomal degradation.

Belogurov A Jr, Kudriaeva A, Kuzina E, Smirnov I, Bobik T, Ponomarenko N, Kravtsova-Ivantsiv Y, Ciechanover A, Gabibov A. Belogurov A Jr, et al. J Biol Chem. 2014 Jun 20;289(25):17758-66. doi: 10.1074/jbc.M113.544247. Epub 2014 Apr 16. J Biol Chem. 2014. PMID: 24739384 Free PMC article.

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