Humphries SE - Search Results

1

The association of combined alpha and beta fibrinogen genotype on plasma fibrinogen levels in smokers and non-smokers.

Thomas AE, Green FR, Lamlum H, Humphries SE. Thomas AE, et al. Among authors: humphries se. J Med Genet. 1995 Aug;32(8):585-9. doi: 10.1136/jmg.32.8.585. J Med Genet. 1995. PMID: 7473646 Free PMC article.

2

Variation in the promoter region of the beta fibrinogen gene is associated with plasma fibrinogen levels in smokers and non-smokers.

Thomas AE, Green FR, Kelleher CH, Wilkes HC, Brennan PJ, Meade TW, Humphries SE. Thomas AE, et al. Among authors: humphries se. Thromb Haemost. 1991 May 6;65(5):487-90. Thromb Haemost. 1991. PMID: 1871708

3

Identification of genetic variation that determines levels of plasma triglycerides and hypercoagulability.

Humphries SE, Peacock R, Dunning A, Lane A, Green F, Hamsten A. Humphries SE, et al. Clin Genet. 1994 Jul;46(1 Spec No):19-31. doi: 10.1111/j.1399-0004.1994.tb04198.x. Clin Genet. 1994. PMID: 7988074 Review. No abstract available.

4

Factor VII coagulant activity and antigen levels in healthy men are determined by interaction between factor VII genotype and plasma triglyceride concentration.

Humphries SE, Lane A, Green FR, Cooper J, Miller GJ. Humphries SE, et al. Arterioscler Thromb. 1994 Feb;14(2):193-8. doi: 10.1161/01.atv.14.2.193. Arterioscler Thromb. 1994. PMID: 8305408

5

Association of genetic variation at the beta-fibrinogen gene locus and plasma fibrinogen levels; interaction between allele frequency of the G/A-455 polymorphism, age and smoking.

Thomas AE, Green FR, Humphries SE. Thomas AE, et al. Among authors: humphries se. Clin Genet. 1996 Oct;50(4):184-90. doi: 10.1111/j.1399-0004.1996.tb02623.x. Clin Genet. 1996. PMID: 9001796

6

The study of gene-environment interactions that influence thrombosis and fibrinolysis. Genetic variation at the loci for factor VII and plasminogen activator inhibitor-1.

Humphries SE, Lane A, Dawson S, Green FR. Humphries SE, et al. Arch Pathol Lab Med. 1992 Dec;116(12):1322-9. Arch Pathol Lab Med. 1992. PMID: 1456879 Review.

7

Apolipoprotein (apo) E genotypes by polymerase chain reaction and allele-specific oligonucleotide probes: no detectable linkage disequilibrium between apo E and apo CII.

Houlston RS, Snowden C, Green F, Alberti KG, Humphries SE. Houlston RS, et al. Among authors: humphries se. Hum Genet. 1989 Nov;83(4):364-8. doi: 10.1007/BF00291382. Hum Genet. 1989. PMID: 2572535

8

Determinants of plasminogen activator inhibitor 1 activity in treated NIDDM and its relation to a polymorphism in the plasminogen activator inhibitor 1 gene.

Panahloo A, Mohamed-Ali V, Lane A, Green F, Humphries SE, Yudkin JS. Panahloo A, et al. Among authors: humphries se. Diabetes. 1995 Jan;44(1):37-42. doi: 10.2337/diab.44.1.37. Diabetes. 1995. PMID: 7813812 Clinical Trial.

9

The 4G/5G genetic polymorphism in the promoter of the plasminogen activator inhibitor-1 (PAI-1) gene is associated with differences in plasma PAI-1 activity but not with risk of myocardial infarction in the ECTIM study. Etude CasTemoins de I'nfarctus du Mycocarde.

Ye S, Green FR, Scarabin PY, Nicaud V, Bara L, Dawson SJ, Humphries SE, Evans A, Luc G, Cambou JP, et al. Ye S, et al. Among authors: humphries se. Thromb Haemost. 1995 Sep;74(3):837-41. Thromb Haemost. 1995. PMID: 8571307

10

Gene-environment interaction in the determination of levels of haemostatic variables involved in thrombosis and fibrinolysis.

Humphries SE, Panahloo A, Montgomery HE, Green F, Yudkin J. Humphries SE, et al. Thromb Haemost. 1997 Jul;78(1):457-61. Thromb Haemost. 1997. PMID: 9198196 Review.

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