Kosaka H - Search Results

1

Beneficial effects of inhibition of angiotensin-converting enzyme on ischemic myocardium during coronary hypoperfusion in dogs.

Kitakaze M, Minamino T, Node K, Komamura K, Shinozaki Y, Mori H, Kosaka H, Inoue M, Hori M, Kamada T. Kitakaze M, et al. Among authors: kosaka h. Circulation. 1995 Aug 15;92(4):950-61. doi: 10.1161/01.cir.92.4.950. Circulation. 1995. PMID: 7641379

2

Bradykinin mediation of Ca(2+)-activated K+ channels regulates coronary blood flow in ischemic myocardium.

Node K, Kitakaze M, Kosaka H, Minamino T, Hori M. Node K, et al. Among authors: kosaka h. Circulation. 1997 Mar 18;95(6):1560-7. doi: 10.1161/01.cir.95.6.1560. Circulation. 1997. PMID: 9118526

3

Roles of NO and Ca2+-activated K+ channels in coronary vasodilation induced by 17beta-estradiol in ischemic heart failure.

Node K, Kitakaze M, Kosaka H, Minamino T, Sato H, Kuzuya T, Hori M. Node K, et al. Among authors: kosaka h. FASEB J. 1997 Aug;11(10):793-9. doi: 10.1096/fasebj.11.10.9271364. FASEB J. 1997. PMID: 9271364

4

Amelioration of ischemia- and reperfusion-induced myocardial injury by 17beta-estradiol: role of nitric oxide and calcium-activated potassium channels.

Node K, Kitakaze M, Kosaka H, Minamino T, Funaya H, Hori M. Node K, et al. Among authors: kosaka h. Circulation. 1997 Sep 16;96(6):1953-63. doi: 10.1161/01.cir.96.6.1953. Circulation. 1997. PMID: 9323086

5

Role of Ca2+-activated K+ channels in the protective effect of ACE inhibition against ischemic myocardial injury.

Node K, Kitakaze M, Kosaka H, Minamino T, Mori H, Hori M. Node K, et al. Among authors: kosaka h. Hypertension. 1998 Jun;31(6):1290-8. doi: 10.1161/01.hyp.31.6.1290. Hypertension. 1998. PMID: 9622144

6

Inhibition of angiotensin-converting enzyme increases the nitric oxide levels in canine ischemic myocardium.

Kitakaze M, Node K, Minamino T, Asanuma H, Ueda Y, Kosaka H, Kuzuya T, Hori M. Kitakaze M, et al. Among authors: kosaka h. J Mol Cell Cardiol. 1998 Nov;30(11):2461-6. doi: 10.1006/jmcc.1998.0806. J Mol Cell Cardiol. 1998. PMID: 9925380

7

Intracoronary administration of adenosine triphosphate increases coronary blood flow and attenuates the severity of myocardial ischemic injury in dogs.

Kitakaze M, Node K, Komamura K, Minamino T, Kosaka H, Kuzuya T, Hori M. Kitakaze M, et al. Among authors: kosaka h. Cardiovasc Drugs Ther. 1999 Sep;13(5):407-14. doi: 10.1023/a:1007899822136. Cardiovasc Drugs Ther. 1999. PMID: 10547220

8

Roles of alpha 1-adrenoceptor activity in the release of nitric oxide during ischemia of the canine heart.

Node K, Kitakaze M, Kosaka H, Komamura K, Minamino T, Tada M, Inoue M, Hori M, Kamada T. Node K, et al. Among authors: kosaka h. Biochem Biophys Res Commun. 1995 Jul 26;212(3):1133-8. doi: 10.1006/bbrc.1995.2086. Biochem Biophys Res Commun. 1995. PMID: 7626102

9

Plasma nitric oxide end products are increased in the ischemic canine heart.

Node K, Kitakaze M, Kosaka H, Komamura K, Minamino T, Tada M, Inoue M, Hori M, Kamada T. Node K, et al. Among authors: kosaka h. Biochem Biophys Res Commun. 1995 Jun 15;211(2):370-4. doi: 10.1006/bbrc.1995.1823. Biochem Biophys Res Commun. 1995. PMID: 7794246

10

Increased release of NO during ischemia reduces myocardial contractility and improves metabolic dysfunction.

Node K, Kitakaze M, Kosaka H, Komamura K, Minamino T, Inoue M, Tada M, Hori M, Kamada T. Node K, et al. Among authors: kosaka h. Circulation. 1996 Jan 15;93(2):356-64. doi: 10.1161/01.cir.93.2.356. Circulation. 1996. PMID: 8548910

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