Mehta A - Search Results

1

In situ DNA PCR and RNA hybridization detection of herpes simplex virus sequences in trigeminal ganglia of latently infected mice.

Mehta A, Maggioncalda J, Bagasra O, Thikkavarapu S, Saikumari P, Valyi-Nagy T, Fraser NW, Block TM. Mehta A, et al. Virology. 1995 Jan 10;206(1):633-40. doi: 10.1016/s0042-6822(95)80080-8. Virology. 1995. PMID: 7831818 Free article.

2

Analysis of a herpes simplex virus type 1 LAT mutant with a deletion between the putative promoter and the 5' end of the 2.0-kilobase transcript.

Maggioncalda J, Mehta A, Fraser NW, Block TM. Maggioncalda J, et al. Among authors: mehta a. J Virol. 1994 Dec;68(12):7816-24. doi: 10.1128/JVI.68.12.7816-7824.1994. J Virol. 1994. PMID: 7966571 Free PMC article.

3

A herpes simplex virus type 1 mutant with a deletion immediately upstream of the LAT locus establishes latency and reactivates from latently infected mice with normal kinetics.

Maggioncalda J, Mehta A, Bagasra O, Fraser NW, Block TM. Maggioncalda J, et al. Among authors: mehta a. J Neurovirol. 1996 Aug;2(4):268-78. doi: 10.3109/13550289609146890. J Neurovirol. 1996. PMID: 8799218

4

Correlation between herpes simplex virus type 1 rate of reactivation from latent infection and the number of infected neurons in trigeminal ganglia.

Maggioncalda J, Mehta A, Su YH, Fraser NW, Block TM. Maggioncalda J, et al. Among authors: mehta a. Virology. 1996 Nov 1;225(1):72-81. doi: 10.1006/viro.1996.0576. Virology. 1996. PMID: 8918535 Free article.

5

The HSV 1 genome in quiescently infected NGF differentiated PC12 cells can not be stimulated by HSV superinfection.

Su YH, Moxley M, Kejariwal R, Mehta A, Fraser NW, Block TM. Su YH, et al. Among authors: mehta a. J Neurovirol. 2000 Aug;6(4):341-9. doi: 10.3109/13550280009030760. J Neurovirol. 2000. PMID: 10951558

6

Hepatitis B virus-mediated changes of apolipoprotein mRNA abundance in cultured hepatoma cells.

Norton PA, Gong Q, Mehta AS, Lu X, Block TM. Norton PA, et al. Among authors: mehta as. J Virol. 2003 May;77(9):5503-6. doi: 10.1128/jvi.77.9.5503-5506.2003. J Virol. 2003. PMID: 12692252 Free PMC article.

7

Inhibition of host ER glucosidase activity prevents Golgi processing of virion-associated bovine viral diarrhea virus E2 glycoproteins and reduces infectivity of secreted virions.

Jordan R, Nikolaeva OV, Wang L, Conyers B, Mehta A, Dwek RA, Block TM. Jordan R, et al. Among authors: mehta a. Virology. 2002 Mar 30;295(1):10-9. doi: 10.1006/viro.2002.1370. Virology. 2002. PMID: 12033761 Free article.

8

Hepatitis B virus large and middle glycoproteins are degraded by a proteasome pathway in glucosidase-inhibited cells but not in cells with functional glucosidase enzyme.

Simsek E, Mehta A, Zhou T, Dwek RA, Block T. Simsek E, et al. Among authors: mehta a. J Virol. 2005 Oct;79(20):12914-20. doi: 10.1128/JVI.79.20.12914-12920.2005. J Virol. 2005. PMID: 16188993 Free PMC article.

9

Evidence that N-linked glycosylation is necessary for hepatitis B virus secretion.

Lu X, Mehta A, Dwek R, Butters T, Block T. Lu X, et al. Among authors: mehta a. Virology. 1995 Nov 10;213(2):660-5. doi: 10.1006/viro.1995.0038. Virology. 1995. PMID: 7491790 Free article.

10

Aberrant trafficking of hepatitis B virus glycoproteins in cells in which N-glycan processing is inhibited.

Lu X, Mehta A, Dadmarz M, Dwek R, Blumberg BS, Block TM. Lu X, et al. Among authors: mehta a. Proc Natl Acad Sci U S A. 1997 Mar 18;94(6):2380-5. doi: 10.1073/pnas.94.6.2380. Proc Natl Acad Sci U S A. 1997. PMID: 9122203 Free PMC article.

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