Ohta S - Search Results

1

Inherited amyloid polyneuropathy type IV (gelsolin variant) in a Japanese family.

Sunada Y, Shimizu T, Nakase H, Ohta S, Asaoka T, Amano S, Sawa M, Kagawa Y, Kanazawa I, Mannen T. Sunada Y, et al. Among authors: ohta s. Ann Neurol. 1993 Jan;33(1):57-62. doi: 10.1002/ana.410330110. Ann Neurol. 1993. PMID: 8388189

2

Widespread tissue distribution of multiple mitochondrial DNA deletions in familial mitochondrial myopathy.

Kawashima S, Ohta S, Kagawa Y, Yoshida M, Nishizawa M. Kawashima S, et al. Among authors: ohta s. Muscle Nerve. 1994 Jul;17(7):741-6. doi: 10.1002/mus.880170707. Muscle Nerve. 1994. PMID: 8008000

3

Multiple deletions in mitochondrial DNA at direct repeats of non-D-loop regions in cases of familial mitochondrial myopathy.

Yuzaki M, Ohkoshi N, Kanazawa I, Kagawa Y, Ohta S. Yuzaki M, et al. Among authors: ohta s. Biochem Biophys Res Commun. 1989 Nov 15;164(3):1352-7. doi: 10.1016/0006-291x(89)91818-4. Biochem Biophys Res Commun. 1989. PMID: 2590206

4

Cytochrome c oxidase--deficient myogenic cell lines in mitochondrial myopathy.

Shimoizumi H, Momoi MY, Ohta S, Kagawa Y, Momoi T, Yanagisawa M. Shimoizumi H, et al. Among authors: ohta s. Ann Neurol. 1989 Jun;25(6):615-21. doi: 10.1002/ana.410250614. Ann Neurol. 1989. PMID: 2545162

5

Content of mutant mitochondrial DNA and organ dysfunction in a patient with a MELAS subgroup of mitochondrial encephalomyopathies.

Shiraiwa N, Ishii A, Iwamoto H, Mizusawa H, Kagawa Y, Ohta S. Shiraiwa N, et al. Among authors: ohta s. J Neurol Sci. 1993 Dec 15;120(2):174-9. doi: 10.1016/0022-510x(93)90270-9. J Neurol Sci. 1993. PMID: 8138807

6

Smaller isoform of human mitochondrial transcription factor 1: its wide distribution and production by alternative splicing.

Tominaga K, Hayashi J, Kagawa Y, Ohta S. Tominaga K, et al. Among authors: ohta s. Biochem Biophys Res Commun. 1993 Jul 15;194(1):544-51. doi: 10.1006/bbrc.1993.1854. Biochem Biophys Res Commun. 1993. PMID: 8333869

7

Regulation of mitochondrial ATP synthesis in mammalian cells by transcriptional control.

Kagawa Y, Ohta S. Kagawa Y, et al. Among authors: ohta s. Int J Biochem. 1990;22(3):219-29. doi: 10.1016/0020-711x(90)90333-x. Int J Biochem. 1990. PMID: 2185063 Review.

8

Marked decrease of mitochondrial DNA with multiple deletions in a patient with familial mitochondrial myopathy.

Otsuka M, Niijima K, Mizuno Y, Yoshida M, Kagawa Y, Ohta S. Otsuka M, et al. Among authors: ohta s. Biochem Biophys Res Commun. 1990 Mar 16;167(2):680-5. doi: 10.1016/0006-291x(90)92079-f. Biochem Biophys Res Commun. 1990. PMID: 2322247

9

Respiration-deficient cells are caused by a single point mutation in the mitochondrial tRNA-Leu (UUR) gene in mitochondrial myopathy, encephalopathy, lactic acidosis, and strokelike episodes (MELAS).

Kobayashi Y, Momoi MY, Tominaga K, Shimoizumi H, Nihei K, Yanagisawa M, Kagawa Y, Ohta S. Kobayashi Y, et al. Among authors: ohta s. Am J Hum Genet. 1991 Sep;49(3):590-9. Am J Hum Genet. 1991. PMID: 1715668 Free PMC article.

10

The alpha 3 beta 3 and alpha 1 beta 1 complexes of ATP synthase.

Kagawa Y, Ohta S, Harada M, Sato M, Itoh Y. Kagawa Y, et al. Among authors: ohta s. Ann N Y Acad Sci. 1992 Nov 30;671:366-76. doi: 10.1111/j.1749-6632.1992.tb43810.x. Ann N Y Acad Sci. 1992. PMID: 1288333 Review.

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