Dumont J - Search Results

1

Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascades.

Parma J, Van Sande J, Swillens S, Tonacchera M, Dumont J, Vassart G. Parma J, et al. Among authors: dumont j. Mol Endocrinol. 1995 Jun;9(6):725-33. doi: 10.1210/mend.9.6.8592518. Mol Endocrinol. 1995. PMID: 8592518

2

Reciprocal negative regulation between thyrotropin/3',5'-cyclic adenosine monophosphate-mediated proliferation and caveolin-1 expression in human and murine thyrocytes.

Costa MJ, Senou M, Van Rode F, Ruf J, Capello M, Dequanter D, Lothaire P, Dessy C, Dumont JE, Many MC, Van Sande J. Costa MJ, et al. Among authors: dumont je. Mol Endocrinol. 2007 Apr;21(4):921-32. doi: 10.1210/me.2006-0328. Epub 2007 Jan 3. Mol Endocrinol. 2007. PMID: 17202321

3

Somatic and germline mutations of the TSH receptor gene in thyroid diseases.

Van Sande J, Parma J, Tonacchera M, Swillens S, Dumont J, Vassart G. Van Sande J, et al. Among authors: dumont j. J Clin Endocrinol Metab. 1995 Sep;80(9):2577-85. doi: 10.1210/jcem.80.9.7673398. J Clin Endocrinol Metab. 1995. PMID: 7673398 Review. No abstract available.

4

Germline mutations in the thyrotropin receptor gene cause non-autoimmune autosomal dominant hyperthyroidism.

Duprez L, Parma J, Van Sande J, Allgeier A, Leclère J, Schvartz C, Delisle MJ, Decoulx M, Orgiazzi J, Dumont J, et al. Duprez L, et al. Among authors: dumont j. Nat Genet. 1994 Jul;7(3):396-401. doi: 10.1038/ng0794-396. Nat Genet. 1994. PMID: 7920658

5

Constitutively active receptors as a disease-causing mechanism.

Parma J, Duprez L, Van Sande J, Paschke R, Tonacchera M, Dumont J, Vassart G. Parma J, et al. Among authors: dumont j. Mol Cell Endocrinol. 1994 Apr;100(1-2):159-62. doi: 10.1016/0303-7207(94)90296-8. Mol Cell Endocrinol. 1994. PMID: 8056151 Review.

6

Molecular genetics of thyroid diseases.

Ledent C, Parma J, Dumont J, Vassart G, Targovnik H. Ledent C, et al. Among authors: dumont j. Eur J Endocrinol. 1994 Jan;130(1):8-14. doi: 10.1530/eje.0.1300008. Eur J Endocrinol. 1994. PMID: 8124482 Review.

7

Somatic mutations in the thyrotropin receptor gene cause hyperfunctioning thyroid adenomas.

Parma J, Duprez L, Van Sande J, Cochaux P, Gervy C, Mockel J, Dumont J, Vassart G. Parma J, et al. Among authors: dumont j. Nature. 1993 Oct 14;365(6447):649-51. doi: 10.1038/365649a0. Nature. 1993. PMID: 8413627

8

Activating mutations of the TSH receptor gene cause thyroid diseases.

Vassart G, Van Sande J, Parma J, Tonacchera M, Duprez L, Swillens S, Dumont J. Vassart G, et al. Among authors: dumont j. Ann Endocrinol (Paris). 1996;57(1):50-4. Ann Endocrinol (Paris). 1996. PMID: 8734289 Review.

9

In Chinese hamster ovary K1 cells dog and human thyrotropin receptors activate both the cyclic AMP and the phosphatidylinositol 4,5-bisphosphate cascades in the presence of thyrotropin and the cyclic AMP cascade in its absence.

Van Sande J, Swillens S, Gerard C, Allgeier A, Massart C, Vassart G, Dumont JE. Van Sande J, et al. Among authors: dumont je. Eur J Biochem. 1995 Apr 15;229(2):338-43. Eur J Biochem. 1995. PMID: 7744056 Free article.

10

Role of the cyclic adenosine 3',5'-monophosphate and the phosphatidylinositol-Ca2+ cascades in mediating the effects of thyrotropin and iodide on hormone synthesis and secretion in human thyroid slices.

Corvilain B, Laurent E, Lecomte M, Vansande J, Dumont JE. Corvilain B, et al. Among authors: dumont je. J Clin Endocrinol Metab. 1994 Jul;79(1):152-9. doi: 10.1210/jcem.79.1.8027219. J Clin Endocrinol Metab. 1994. PMID: 8027219

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