Schwab R - Search Results

1

Cationic lipid mediated transfer of c-abl and bcr antisense oligonucleotides to immature normal myeloid cells: uptake, biological effects and modulation of gene expression.

Albrecht T, Schwab R, Peschel C, Engels HJ, Fischer T, Huber C, Aulitzky WE. Albrecht T, et al. Among authors: schwab r. Ann Hematol. 1996 Feb;72(2):73-9. doi: 10.1007/BF00641311. Ann Hematol. 1996. PMID: 8597610

2

Primary proliferating immature myeloid cells from CML patients are not resistant to induction of apoptosis by DNA damage and growth factor withdrawal.

Albrecht T, Schwab R, Henkes M, Peschel C, Huber C, Aulitzky WE. Albrecht T, et al. Among authors: schwab r. Br J Haematol. 1996 Dec;95(3):501-7. doi: 10.1046/j.1365-2141.1996.d01-1934.x. Br J Haematol. 1996. PMID: 8943891

3

Deficient cell cycle control in myeloid cells of patients with newly diagnosed chronic myeloid leukemia.

Schwab R, Peschel C, Desprès D, Derigs G, Fischer T, Huber C, Aulitzky WE. Schwab R, et al. Cytokines Mol Ther. 1995 Dec;1(4):281-8. Cytokines Mol Ther. 1995. PMID: 9384681

4

Bcr-Abl kinase promotes cell cycle entry of primary myeloid CML cells in the absence of growth factors.

Jonuleit T, Peschel C, Schwab R, van der Kuip H, Buchdunger E, Fischer T, Huber C, Aulitzky WE. Jonuleit T, et al. Among authors: schwab r. Br J Haematol. 1998 Feb;100(2):295-303. doi: 10.1046/j.1365-2141.1998.00564.x. Br J Haematol. 1998. PMID: 9488616 Free article.

5

Constitutive activation of STAT3 and STAT5 is induced by leukemic fusion proteins with protein tyrosine kinase activity and is sufficient for transformation of hematopoietic precursor cells.

Spiekermann K, Pau M, Schwab R, Schmieja K, Franzrahe S, Hiddemann W. Spiekermann K, et al. Among authors: schwab r. Exp Hematol. 2002 Mar;30(3):262-71. doi: 10.1016/s0301-472x(01)00787-1. Exp Hematol. 2002. PMID: 11882364 Free article.

6

The protein tyrosine kinase inhibitor SU5614 inhibits FLT3 and induces growth arrest and apoptosis in AML-derived cell lines expressing a constitutively activated FLT3.

Spiekermann K, Dirschinger RJ, Schwab R, Bagrintseva K, Faber F, Buske C, Schnittger S, Kelly LM, Gilliland DG, Hiddemann W. Spiekermann K, et al. Among authors: schwab r. Blood. 2003 Feb 15;101(4):1494-504. doi: 10.1182/blood-2002-04-1045. Epub 2002 Oct 24. Blood. 2003. PMID: 12406902 Free article.

7

Overexpression and constitutive activation of FLT3 induces STAT5 activation in primary acute myeloid leukemia blast cells.

Spiekermann K, Bagrintseva K, Schwab R, Schmieja K, Hiddemann W. Spiekermann K, et al. Among authors: schwab r. Clin Cancer Res. 2003 Jun;9(6):2140-50. Clin Cancer Res. 2003. PMID: 12796379

8

Mutations in the tyrosine kinase domain of FLT3 define a new molecular mechanism of acquired drug resistance to PTK inhibitors in FLT3-ITD-transformed hematopoietic cells.

Bagrintseva K, Schwab R, Kohl TM, Schnittger S, Eichenlaub S, Ellwart JW, Hiddemann W, Spiekermann K. Bagrintseva K, et al. Among authors: schwab r. Blood. 2004 Mar 15;103(6):2266-75. doi: 10.1182/blood-2003-05-1653. Epub 2003 Nov 6. Blood. 2004. PMID: 14604974 Free article.

9

How to improve the uterotomy healing. I. Effects of fibrin and tumor necrosis factor-alpha in the rat uterotomy model.

Schlenger K, Höckel M, Schwab R, Frischmann-Berger R, Vaupel P. Schlenger K, et al. Among authors: schwab r. J Surg Res. 1994 Mar;56(3):235-41. doi: 10.1006/jsre.1994.1037. J Surg Res. 1994. PMID: 8145540

10

Liquid biopsy in organ damage: introduction of an innovative tool for assessing the complexity of blunt splenic injury.

Wöhler A, Wang B, Schwab R, Lukacs-Kornek V, Willms AG, Kornek MT. Wöhler A, et al. Among authors: schwab r. Eur J Med Res. 2025 Jan 4;30(1):6. doi: 10.1186/s40001-024-02254-z. Eur J Med Res. 2025. PMID: 39754218 Free PMC article.

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