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Activation of phosphatidylinositol 3-kinase by interleukin-13. An inhibitory signal for inducible nitric-oxide synthase expression in epithelial cell line HT-29.
Wright K, Ward SG, Kolios G, Westwick J. Wright K, et al. Among authors: kolios g. J Biol Chem. 1997 May 9;272(19):12626-33. doi: 10.1074/jbc.272.19.12626. J Biol Chem. 1997. PMID: 9139718 Free article.
The human colonic epithelial cell line HT-29 can be induced by a combination of the cytokines interleukin (IL)-1alpha, tumor necrosis factor alpha, and interferon-gamma to express the inducible form of nitric-oxide synthase (iNOS; Kolios, G., Brown, Z., Robson, R., …
The human colonic epithelial cell line HT-29 can be induced by a combination of the cytokines interleukin (IL)-1alpha, tumor necrosis factor …
Cytokine-induced apoptosis in epithelial HT-29 cells is independent of nitric oxide formation. Evidence for an interleukin-13-driven phosphatidylinositol 3-kinase-dependent survival mechanism.
Wright K, Kolios G, Westwick J, Ward SG. Wright K, et al. Among authors: kolios g. J Biol Chem. 1999 Jun 11;274(24):17193-201. doi: 10.1074/jbc.274.24.17193. J Biol Chem. 1999. PMID: 10358077 Free article.
This can be inhibited by pretreatment with IL-13 via a phosphatidylinositol (PI) 3-kinase-dependent mechanism (Wright, K., Ward, S. G., Kolios, G., and Westwick, J. (1997) J. Biol. Chem. 272, 12626-12633). Since NO has been implicated in regulating mechanisms …
This can be inhibited by pretreatment with IL-13 via a phosphatidylinositol (PI) 3-kinase-dependent mechanism (Wright, K., Ward, S. G
204 results