Greenhalgh DA - Search Results

1

Expression of a dominant-negative type II transforming growth factor beta (TGF-beta) receptor in the epidermis of transgenic mice blocks TGF-beta-mediated growth inhibition.

Wang XJ, Greenhalgh DA, Bickenbach JR, Jiang A, Bundman DS, Krieg T, Derynck R, Roop DR. Wang XJ, et al. Among authors: greenhalgh da. Proc Natl Acad Sci U S A. 1997 Mar 18;94(6):2386-91. doi: 10.1073/pnas.94.6.2386. Proc Natl Acad Sci U S A. 1997. PMID: 9122204 Free PMC article.

2

TGF alpha and v-fos cooperation in transgenic mouse epidermis induces aberrant keratinocyte differentiation and stable, autonomous papillomas.

Wang XJ, Greenhalgh DA, Lu XR, Bickenbach JR, Roop DR. Wang XJ, et al. Among authors: greenhalgh da. Oncogene. 1995 Jan 19;10(2):279-89. Oncogene. 1995. PMID: 7530825

3

Hyperplasia, hyperkeratosis and benign tumor production in transgenic mice by a targeted v-fos oncogene suggest a role for fos in epidermal differentiation and neoplasia.

Greenhalgh DA, Rothnagel JA, Wang XJ, Quintanilla MI, Orengo CC, Gagne TA, Bundman DS, Longley MA, Fisher C, Roop DR. Greenhalgh DA, et al. Oncogene. 1993 Aug;8(8):2145-57. Oncogene. 1993. PMID: 7687760

4

Transgenic models of skin diseases.

Rothnagel JA, Greenhalgh DA, Wang XJ, Sellheyer K, Bickenbach JR, Dominey AM, Roop DR. Rothnagel JA, et al. Among authors: greenhalgh da. Arch Dermatol. 1993 Nov;129(11):1430-6. Arch Dermatol. 1993. PMID: 7694550 Free article. Review.

5

Targeted overexpression of transforming growth factor alpha in the epidermis of transgenic mice elicits hyperplasia, hyperkeratosis, and spontaneous, squamous papillomas.

Dominey AM, Wang XJ, King LE Jr, Nanney LB, Gagne TA, Sellheyer K, Bundman DS, Longley MA, Rothnagel JA, Greenhalgh DA, et al. Dominey AM, et al. Among authors: greenhalgh da. Cell Growth Differ. 1993 Dec;4(12):1071-82. Cell Growth Differ. 1993. PMID: 8117621

6

Epidermal expression of transforming growth factor-alpha in transgenic mice: induction of spontaneous and 12-O-tetradecanoylphorbol-13-acetate-induced papillomas via a mechanism independent of Ha-ras activation or overexpression.

Wang XJ, Greenhalgh DA, Eckhardt JN, Rothnagel JA, Roop DR. Wang XJ, et al. Among authors: greenhalgh da. Mol Carcinog. 1994 May;10(1):15-22. doi: 10.1002/mc.2940100104. Mol Carcinog. 1994. PMID: 8185825

7

Multistage skin carcinogenesis in transgenic mice.

Greenhalgh DA, Wang XJ, Roop DR. Greenhalgh DA, et al. Proc Assoc Am Physicians. 1995 Jul;107(2):258-75. Proc Assoc Am Physicians. 1995. PMID: 8624862 Review. No abstract available.

8

Paradoxical tumor inhibitory effect of p53 loss in transgenic mice expressing epidermal-targeted v-rasHa, v-fos, or human transforming growth factor alpha.

Greenhalgh DA, Wang XJ, Donehower LA, Roop DR. Greenhalgh DA, et al. Cancer Res. 1996 Oct 1;56(19):4413-23. Cancer Res. 1996. PMID: 8813135

9

Multistage epidermal carcinogenesis in transgenic mice: cooperativity and paradox.

Greenhalgh DA, Wang XJ, Roop DR. Greenhalgh DA, et al. J Investig Dermatol Symp Proc. 1996 Apr;1(2):162-76. J Investig Dermatol Symp Proc. 1996. PMID: 9627712 Review.

10

Expression of a p53 mutant in the epidermis of transgenic mice accelerates chemical carcinogenesis.

Wang XJ, Greenhalgh DA, Jiang A, He D, Zhong L, Medina D, Brinkley BR, Roop DR. Wang XJ, et al. Among authors: greenhalgh da. Oncogene. 1998 Jul 9;17(1):35-45. doi: 10.1038/sj.onc.1201890. Oncogene. 1998. PMID: 9671312

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